|
Inhibition of Oryctolagus cuniculus (rabbit) AOX in liver cytosol at IC50 concentration
|
Oryctolagus cuniculus
|
-2.0
%
|
|
Journal : J Agric Food Chem
Title : Neonicotinoid insecticides: oxidative stress in planta and metallo-oxidase inhibition.
Year : 2011
Volume : 59
Issue : 9
First Page : 4860
Last Page : 4867
Authors : Ford KA, Gulevich AG, Swenson TL, Casida JE.
Abstract : Neonicotinoids not only control insect pests but also sometimes independently alter plant growth and response to stress. We find that imidacloprid, thiacloprid, acetamiprid, thiamethoxam, and clothianidin but not nitenpyram and dinotefuran induce foliar lesions and peroxidative damage in soybean ( Glycine max ) seedlings assayed with the 3,3'-diaminobenzidine stain. The chloropyridinyl-carboxylic acid (COOH) but not the -carboxaldehyde (CHO) metabolites induce peroxidative damage but in a different pattern. Surprisingly, the chlorothiazolyl -CHO and -COOH metabolites induce chlorosis but no clear superimposable peroxidative damage or cell death. Four metallo-oxidases known to modulate reactive oxygen species were not sensitive in vitro to the parent neonicotinoid itself but were to several CHO and COOH metabolites and related compounds, with a sensitivity order of CHO > COOH and tyrosinase > xanthine oxidase and aldehyde oxidase > catalase. Although metallo-oxidase inhibition does not correlate overall with lesion formation, it may play an as yet unknown role in plant response to neonicotinoids.
|
Inhibition of xanthine oxidase at IC50 concentration
|
None
|
3.0
%
|
|
Journal : J Agric Food Chem
Title : Neonicotinoid insecticides: oxidative stress in planta and metallo-oxidase inhibition.
Year : 2011
Volume : 59
Issue : 9
First Page : 4860
Last Page : 4867
Authors : Ford KA, Gulevich AG, Swenson TL, Casida JE.
Abstract : Neonicotinoids not only control insect pests but also sometimes independently alter plant growth and response to stress. We find that imidacloprid, thiacloprid, acetamiprid, thiamethoxam, and clothianidin but not nitenpyram and dinotefuran induce foliar lesions and peroxidative damage in soybean ( Glycine max ) seedlings assayed with the 3,3'-diaminobenzidine stain. The chloropyridinyl-carboxylic acid (COOH) but not the -carboxaldehyde (CHO) metabolites induce peroxidative damage but in a different pattern. Surprisingly, the chlorothiazolyl -CHO and -COOH metabolites induce chlorosis but no clear superimposable peroxidative damage or cell death. Four metallo-oxidases known to modulate reactive oxygen species were not sensitive in vitro to the parent neonicotinoid itself but were to several CHO and COOH metabolites and related compounds, with a sensitivity order of CHO > COOH and tyrosinase > xanthine oxidase and aldehyde oxidase > catalase. Although metallo-oxidase inhibition does not correlate overall with lesion formation, it may play an as yet unknown role in plant response to neonicotinoids.
|
Inhibition of xanthine oxidase
|
None
|
1000000.0
nM
|
|
Journal : J Agric Food Chem
Title : Neonicotinoid insecticides: oxidative stress in planta and metallo-oxidase inhibition.
Year : 2011
Volume : 59
Issue : 9
First Page : 4860
Last Page : 4867
Authors : Ford KA, Gulevich AG, Swenson TL, Casida JE.
Abstract : Neonicotinoids not only control insect pests but also sometimes independently alter plant growth and response to stress. We find that imidacloprid, thiacloprid, acetamiprid, thiamethoxam, and clothianidin but not nitenpyram and dinotefuran induce foliar lesions and peroxidative damage in soybean ( Glycine max ) seedlings assayed with the 3,3'-diaminobenzidine stain. The chloropyridinyl-carboxylic acid (COOH) but not the -carboxaldehyde (CHO) metabolites induce peroxidative damage but in a different pattern. Surprisingly, the chlorothiazolyl -CHO and -COOH metabolites induce chlorosis but no clear superimposable peroxidative damage or cell death. Four metallo-oxidases known to modulate reactive oxygen species were not sensitive in vitro to the parent neonicotinoid itself but were to several CHO and COOH metabolites and related compounds, with a sensitivity order of CHO > COOH and tyrosinase > xanthine oxidase and aldehyde oxidase > catalase. Although metallo-oxidase inhibition does not correlate overall with lesion formation, it may play an as yet unknown role in plant response to neonicotinoids.
|
Inhibition of Oryctolagus cuniculus (rabbit) AOX in liver cytosol
|
Oryctolagus cuniculus
|
1000000.0
nM
|
|
Journal : J Agric Food Chem
Title : Neonicotinoid insecticides: oxidative stress in planta and metallo-oxidase inhibition.
Year : 2011
Volume : 59
Issue : 9
First Page : 4860
Last Page : 4867
Authors : Ford KA, Gulevich AG, Swenson TL, Casida JE.
Abstract : Neonicotinoids not only control insect pests but also sometimes independently alter plant growth and response to stress. We find that imidacloprid, thiacloprid, acetamiprid, thiamethoxam, and clothianidin but not nitenpyram and dinotefuran induce foliar lesions and peroxidative damage in soybean ( Glycine max ) seedlings assayed with the 3,3'-diaminobenzidine stain. The chloropyridinyl-carboxylic acid (COOH) but not the -carboxaldehyde (CHO) metabolites induce peroxidative damage but in a different pattern. Surprisingly, the chlorothiazolyl -CHO and -COOH metabolites induce chlorosis but no clear superimposable peroxidative damage or cell death. Four metallo-oxidases known to modulate reactive oxygen species were not sensitive in vitro to the parent neonicotinoid itself but were to several CHO and COOH metabolites and related compounds, with a sensitivity order of CHO > COOH and tyrosinase > xanthine oxidase and aldehyde oxidase > catalase. Although metallo-oxidase inhibition does not correlate overall with lesion formation, it may play an as yet unknown role in plant response to neonicotinoids.
|
Inhibition of Agaricus bisporus (mushroom) tyrosinase
|
Agaricus bisporus
|
1740000.0
nM
|
|
Journal : J Agric Food Chem
Title : Neonicotinoid insecticides: oxidative stress in planta and metallo-oxidase inhibition.
Year : 2011
Volume : 59
Issue : 9
First Page : 4860
Last Page : 4867
Authors : Ford KA, Gulevich AG, Swenson TL, Casida JE.
Abstract : Neonicotinoids not only control insect pests but also sometimes independently alter plant growth and response to stress. We find that imidacloprid, thiacloprid, acetamiprid, thiamethoxam, and clothianidin but not nitenpyram and dinotefuran induce foliar lesions and peroxidative damage in soybean ( Glycine max ) seedlings assayed with the 3,3'-diaminobenzidine stain. The chloropyridinyl-carboxylic acid (COOH) but not the -carboxaldehyde (CHO) metabolites induce peroxidative damage but in a different pattern. Surprisingly, the chlorothiazolyl -CHO and -COOH metabolites induce chlorosis but no clear superimposable peroxidative damage or cell death. Four metallo-oxidases known to modulate reactive oxygen species were not sensitive in vitro to the parent neonicotinoid itself but were to several CHO and COOH metabolites and related compounds, with a sensitivity order of CHO > COOH and tyrosinase > xanthine oxidase and aldehyde oxidase > catalase. Although metallo-oxidase inhibition does not correlate overall with lesion formation, it may play an as yet unknown role in plant response to neonicotinoids.
|
Phytotoxicity against Vitis vinifera seedlings assessed as lesions at 50 to 100 ppm after 9 days
|
Vitis vinifera
|
None
|
|
Journal : J Agric Food Chem
Title : Neonicotinoid insecticides: oxidative stress in planta and metallo-oxidase inhibition.
Year : 2011
Volume : 59
Issue : 9
First Page : 4860
Last Page : 4867
Authors : Ford KA, Gulevich AG, Swenson TL, Casida JE.
Abstract : Neonicotinoids not only control insect pests but also sometimes independently alter plant growth and response to stress. We find that imidacloprid, thiacloprid, acetamiprid, thiamethoxam, and clothianidin but not nitenpyram and dinotefuran induce foliar lesions and peroxidative damage in soybean ( Glycine max ) seedlings assayed with the 3,3'-diaminobenzidine stain. The chloropyridinyl-carboxylic acid (COOH) but not the -carboxaldehyde (CHO) metabolites induce peroxidative damage but in a different pattern. Surprisingly, the chlorothiazolyl -CHO and -COOH metabolites induce chlorosis but no clear superimposable peroxidative damage or cell death. Four metallo-oxidases known to modulate reactive oxygen species were not sensitive in vitro to the parent neonicotinoid itself but were to several CHO and COOH metabolites and related compounds, with a sensitivity order of CHO > COOH and tyrosinase > xanthine oxidase and aldehyde oxidase > catalase. Although metallo-oxidase inhibition does not correlate overall with lesion formation, it may play an as yet unknown role in plant response to neonicotinoids.
|
Phytotoxicity against Zea mays (maize) seedlings assessed as lesions at 50 to 100 ppm after 9 days
|
Zea mays
|
None
|
|
Journal : J Agric Food Chem
Title : Neonicotinoid insecticides: oxidative stress in planta and metallo-oxidase inhibition.
Year : 2011
Volume : 59
Issue : 9
First Page : 4860
Last Page : 4867
Authors : Ford KA, Gulevich AG, Swenson TL, Casida JE.
Abstract : Neonicotinoids not only control insect pests but also sometimes independently alter plant growth and response to stress. We find that imidacloprid, thiacloprid, acetamiprid, thiamethoxam, and clothianidin but not nitenpyram and dinotefuran induce foliar lesions and peroxidative damage in soybean ( Glycine max ) seedlings assayed with the 3,3'-diaminobenzidine stain. The chloropyridinyl-carboxylic acid (COOH) but not the -carboxaldehyde (CHO) metabolites induce peroxidative damage but in a different pattern. Surprisingly, the chlorothiazolyl -CHO and -COOH metabolites induce chlorosis but no clear superimposable peroxidative damage or cell death. Four metallo-oxidases known to modulate reactive oxygen species were not sensitive in vitro to the parent neonicotinoid itself but were to several CHO and COOH metabolites and related compounds, with a sensitivity order of CHO > COOH and tyrosinase > xanthine oxidase and aldehyde oxidase > catalase. Although metallo-oxidase inhibition does not correlate overall with lesion formation, it may play an as yet unknown role in plant response to neonicotinoids.
|
Phytotoxicity against Gossypium hirsutum (cotton) seedlings assessed as lesions at 50 to 100 ppm after 9 days
|
Gossypium hirsutum
|
None
|
|
Journal : J Agric Food Chem
Title : Neonicotinoid insecticides: oxidative stress in planta and metallo-oxidase inhibition.
Year : 2011
Volume : 59
Issue : 9
First Page : 4860
Last Page : 4867
Authors : Ford KA, Gulevich AG, Swenson TL, Casida JE.
Abstract : Neonicotinoids not only control insect pests but also sometimes independently alter plant growth and response to stress. We find that imidacloprid, thiacloprid, acetamiprid, thiamethoxam, and clothianidin but not nitenpyram and dinotefuran induce foliar lesions and peroxidative damage in soybean ( Glycine max ) seedlings assayed with the 3,3'-diaminobenzidine stain. The chloropyridinyl-carboxylic acid (COOH) but not the -carboxaldehyde (CHO) metabolites induce peroxidative damage but in a different pattern. Surprisingly, the chlorothiazolyl -CHO and -COOH metabolites induce chlorosis but no clear superimposable peroxidative damage or cell death. Four metallo-oxidases known to modulate reactive oxygen species were not sensitive in vitro to the parent neonicotinoid itself but were to several CHO and COOH metabolites and related compounds, with a sensitivity order of CHO > COOH and tyrosinase > xanthine oxidase and aldehyde oxidase > catalase. Although metallo-oxidase inhibition does not correlate overall with lesion formation, it may play an as yet unknown role in plant response to neonicotinoids.
|
Phytotoxicity against Spinacia oleracea (spinach) seedlings assessed as lesions at 100 ppm after 9 days
|
Spinacia oleracea
|
None
|
|
Journal : J Agric Food Chem
Title : Neonicotinoid insecticides: oxidative stress in planta and metallo-oxidase inhibition.
Year : 2011
Volume : 59
Issue : 9
First Page : 4860
Last Page : 4867
Authors : Ford KA, Gulevich AG, Swenson TL, Casida JE.
Abstract : Neonicotinoids not only control insect pests but also sometimes independently alter plant growth and response to stress. We find that imidacloprid, thiacloprid, acetamiprid, thiamethoxam, and clothianidin but not nitenpyram and dinotefuran induce foliar lesions and peroxidative damage in soybean ( Glycine max ) seedlings assayed with the 3,3'-diaminobenzidine stain. The chloropyridinyl-carboxylic acid (COOH) but not the -carboxaldehyde (CHO) metabolites induce peroxidative damage but in a different pattern. Surprisingly, the chlorothiazolyl -CHO and -COOH metabolites induce chlorosis but no clear superimposable peroxidative damage or cell death. Four metallo-oxidases known to modulate reactive oxygen species were not sensitive in vitro to the parent neonicotinoid itself but were to several CHO and COOH metabolites and related compounds, with a sensitivity order of CHO > COOH and tyrosinase > xanthine oxidase and aldehyde oxidase > catalase. Although metallo-oxidase inhibition does not correlate overall with lesion formation, it may play an as yet unknown role in plant response to neonicotinoids.
|
Phytotoxicity against Glycine max (soybean) seedlings assessed as lesions in unfoliolate leaves at 100 ppm after 9 days
|
Glycine max
|
None
|
|
Journal : J Agric Food Chem
Title : Neonicotinoid insecticides: oxidative stress in planta and metallo-oxidase inhibition.
Year : 2011
Volume : 59
Issue : 9
First Page : 4860
Last Page : 4867
Authors : Ford KA, Gulevich AG, Swenson TL, Casida JE.
Abstract : Neonicotinoids not only control insect pests but also sometimes independently alter plant growth and response to stress. We find that imidacloprid, thiacloprid, acetamiprid, thiamethoxam, and clothianidin but not nitenpyram and dinotefuran induce foliar lesions and peroxidative damage in soybean ( Glycine max ) seedlings assayed with the 3,3'-diaminobenzidine stain. The chloropyridinyl-carboxylic acid (COOH) but not the -carboxaldehyde (CHO) metabolites induce peroxidative damage but in a different pattern. Surprisingly, the chlorothiazolyl -CHO and -COOH metabolites induce chlorosis but no clear superimposable peroxidative damage or cell death. Four metallo-oxidases known to modulate reactive oxygen species were not sensitive in vitro to the parent neonicotinoid itself but were to several CHO and COOH metabolites and related compounds, with a sensitivity order of CHO > COOH and tyrosinase > xanthine oxidase and aldehyde oxidase > catalase. Although metallo-oxidase inhibition does not correlate overall with lesion formation, it may play an as yet unknown role in plant response to neonicotinoids.
|
Adulticidal activity against female Bemisia tabaci biotype B (sweet potato whitefly) in cucumber leaves assessed as residual contact toxicity treated for 30 secs before adult insect infestation measured after 24 hr by leaf dip bioassay
|
Bemisia tabaci
|
11.6
ppm
|
|
Journal : J Agric Food Chem
Year : 2011
Volume : 59
Issue : 15
First Page : 8193
Last Page : 8198
|
Biotype susceptibility ratio, ratio of LC50 for female adult Bemisia tabaci biotype Q (sweet potato whitefly) to LC50 for female adult Bemisia tabaci biotype B
|
Bemisia tabaci
|
31.4
|
|
Journal : J Agric Food Chem
Year : 2011
Volume : 59
Issue : 15
First Page : 8193
Last Page : 8198
|
Adulticidal activity against female Bemisia tabaci biotype Q (sweet potato whitefly) in cucumber leaves assessed as residual contact toxicity treated for 30 secs before adult insect infestation measured after 24 hr by leaf dip bioassay
|
Bemisia tabaci
|
364.5
ppm
|
|
Journal : J Agric Food Chem
Year : 2011
Volume : 59
Issue : 15
First Page : 8193
Last Page : 8198
|
Drug metabolism in Drosophila melanogaster D.mel-2 cells over-expressing in CYP6G1 assessed as drug recovery measured 24 hr post dose by HPLC method
|
Drosophila melanogaster
|
0.0
%
|
|
Journal : J Agric Food Chem
Year : 2011
Volume : 59
Issue : 7
First Page : 2950
Last Page : 2957
|
Drug metabolism in Drosophila melanogaster D.mel-2 cells deficient in CYP6G1 assessed as drug recovery measured 24 hr post dose by HPLC method
|
Drosophila melanogaster
|
122.7
%
|
|
Journal : J Agric Food Chem
Year : 2011
Volume : 59
Issue : 7
First Page : 2950
Last Page : 2957
|
Pesticidal activity against Contarinia nasturtii grown on broccoli assessed as larval pupation applied as foliar spray 8 days post-ovipositioning relative to control
|
Contarinia nasturtii
|
20.0
%
|
|
Journal : Crop Protection
Year : 2007
Volume : 26
Issue : 10
First Page : 1574
Last Page : 1578
|
Pesticidal activity against Contarinia nasturtii grown on broccoli assessed as larvae survival applied as foliar spray 8 days post-ovipositioning relative to control
|
Contarinia nasturtii
|
30.0
%
|
|
Journal : Crop Protection
Year : 2007
Volume : 26
Issue : 10
First Page : 1574
Last Page : 1578
|
Pesticidal activity against Contarinia nasturtii grown on broccoli applied as foliar spray immediately post-ovipositioning relative to control
|
Contarinia nasturtii
|
99.9
%
|
|
Journal : Crop Protection
Year : 2007
Volume : 26
Issue : 10
First Page : 1574
Last Page : 1578
|
Pesticidal activity against Contarinia nasturtii grown on three-leaf stage cauliflower seedling at 34 g a.i./ha applied as foliar spray 8 days post-ovipositioning measured after 10 days
|
Contarinia nasturtii
|
69.89
%
|
|
Journal : Crop Protection
Year : 2007
Volume : 26
Issue : 10
First Page : 1574
Last Page : 1578
|
Pesticidal activity against Contarinia nasturtii grown on three-leaf stage cauliflower seedling at 34 g a.i./ha applied as foliar spray 4 days post-ovipositioning measured after 7 days
|
Contarinia nasturtii
|
99.83
%
|
|
Journal : Crop Protection
Year : 2007
Volume : 26
Issue : 10
First Page : 1574
Last Page : 1578
|
Pesticidal activity against Contarinia nasturtii grown on three-leaf stage cauliflower seedling at 34 g a.i./ha applied as foliar spray immediately post-ovipositioning measured after 7 days
|
Contarinia nasturtii
|
100.0
%
|
|
Journal : Crop Protection
Year : 2007
Volume : 26
Issue : 10
First Page : 1574
Last Page : 1578
|
Pesticidal activity against Contarinia nasturtii grown on three-leaf stage cauliflower seedling assessed as decrease in larval feeding time at 34 g a.i./ha applied as foliar spray 8 days post-ovipositioning measured as emergence of adult insect in the soil 7 days after cutting of seedling
|
Contarinia nasturtii
|
None
|
|
Journal : Crop Protection
Year : 2007
Volume : 26
Issue : 10
First Page : 1574
Last Page : 1578
|
Pesticidal activity against Contarinia nasturtii grown on three-leaf stage cauliflower seedling assessed as decrease in mean number of larvae at 34 g a.i./ha applied as foliar spray 8 days post-ovipositioning measured on the above ground part of seedling after 10 days
|
Contarinia nasturtii
|
None
|
|
Journal : Crop Protection
Year : 2007
Volume : 26
Issue : 10
First Page : 1574
Last Page : 1578
|
Pesticidal activity against Contarinia nasturtii grown on three-leaf stage cauliflower seedling assessed as decrease in damage to seedling tips at 34 g a.i./ha applied as foliar spray 8 days post-ovipositioning measured after 10 days
|
Contarinia nasturtii
|
None
|
|
Journal : Crop Protection
Year : 2007
Volume : 26
Issue : 10
First Page : 1574
Last Page : 1578
|
Pesticidal activity against Contarinia nasturtii grown on three-leaf stage cauliflower seedling assessed as insect egg hatching at 34 g a.i./ha applied as foliar spray 8 days post-ovipositioning measured after 10 days (Rvb = 100%)
|
Contarinia nasturtii
|
100.0
%
|
|
Journal : Crop Protection
Year : 2007
Volume : 26
Issue : 10
First Page : 1574
Last Page : 1578
|
Pesticidal activity against Contarinia nasturtii grown on three-leaf stage cauliflower seedling assessed as insect egg hatching at 34 g a.i./ha applied as foliar spray 4 days post-ovipositioning measured after 7 days (Rvb = 99.73 +/- 0.27%)
|
Contarinia nasturtii
|
100.0
%
|
|
Journal : Crop Protection
Year : 2007
Volume : 26
Issue : 10
First Page : 1574
Last Page : 1578
|
Pesticidal activity against Contarinia nasturtii grown on three-leaf stage cauliflower seedling assessed as decrease in insect eggs laid per plant at 34 g a.i./ha applied as foliar spray 4 days post-ovipositioning measured after 7 days relative to control
|
Contarinia nasturtii
|
None
|
|
Journal : Crop Protection
Year : 2007
Volume : 26
Issue : 10
First Page : 1574
Last Page : 1578
|
Pesticidal activity against Contarinia nasturtii grown on three-leaf stage cauliflower seedling assessed as insect egg death at 34 g a.i./ha applied as foliar spray immediately post-ovipositioning measured after 7 days relative to control
|
Contarinia nasturtii
|
100.0
%
|
|
Journal : Crop Protection
Year : 2007
Volume : 26
Issue : 10
First Page : 1574
Last Page : 1578
|
Pesticidal activity against Contarinia nasturtii grown on three-leaf stage cauliflower seedling assessed as insect egg hatching at 34 g a.i./ha applied as foliar spray immediately post-ovipositioning measured after 7 days (Rvb = 100%)
|
Contarinia nasturtii
|
0.0
%
|
|
Journal : Crop Protection
Year : 2007
Volume : 26
Issue : 10
First Page : 1574
Last Page : 1578
|
Pesticidal activity against Contarinia nasturtii grown on three-leaf stage cauliflower seedling assessed as decrease in insect eggs laid per plant at 34 g a.i./ha applied as foliar spray immediately post-ovipositioning measured after 7 days relative to control
|
Contarinia nasturtii
|
None
|
|
Journal : Crop Protection
Year : 2007
Volume : 26
Issue : 10
First Page : 1574
Last Page : 1578
|
Pesticidal activity against Contarinia nasturtii grown on three-leaf stage cauliflower seedling at 34 g a.i./ha applied as foliar spray prior to oviposition measured after 7 days
|
Contarinia nasturtii
|
99.52
%
|
|
Journal : Crop Protection
Year : 2007
Volume : 26
Issue : 10
First Page : 1574
Last Page : 1578
|
Pesticidal activity against Contarinia nasturtii grown on three-leafstage cauliflower seedling assessed as insect egg hatching at 34 g a.i./ha applied as foliar spray prior to oviposition measured after 7 days (Rvb = 100%)
|
Contarinia nasturtii
|
0.48
%
|
|
Journal : Crop Protection
Year : 2007
Volume : 26
Issue : 10
First Page : 1574
Last Page : 1578
|
Pesticidal activity against Contarinia nasturtii grown on three-leaf stage cauliflower seedling assessed as insect egg death at 34 g a.i./ha applied as foliar spray prior to oviposition measured after 7 days
|
Contarinia nasturtii
|
None
|
|
Journal : Crop Protection
Year : 2007
Volume : 26
Issue : 10
First Page : 1574
Last Page : 1578
|
Pesticidal activity against Contarinia nasturtii grown on three-leaf stage cauliflower seedling assessed as decrease in insect eggs laid per plant at 34 g a.i./ha applied as foliar spray prior to oviposition measured after 7 days relative to control
|
Contarinia nasturtii
|
None
|
|
Journal : Crop Protection
Year : 2007
Volume : 26
Issue : 10
First Page : 1574
Last Page : 1578
|
Pesticidal activity against Contarinia nasturtii grown on three-leaf stage cauliflower seedling assessed as mortality of insect adult at 34 g a.i./ha applied as foliar spray prior to oviposition measured after 48 hr relative to control
|
Contarinia nasturtii
|
100.0
%
|
|
Journal : Crop Protection
Year : 2007
Volume : 26
Issue : 10
First Page : 1574
Last Page : 1578
|
Pesticidal activity against Contarinia nasturtii grown on three-leaf stage cauliflower seedling assessed as mortality of insect adult at 34 g a.i./ha applied as foliar spray prior to oviposition measured after 24 hr relative to control
|
Contarinia nasturtii
|
84.44
%
|
|
Journal : Crop Protection
Year : 2007
Volume : 26
Issue : 10
First Page : 1574
Last Page : 1578
|
Insecticidal activity against Bemisia tabaci Bupro-SEL (sweet potato whitefly) from 20 generation buprofezin resistance assessed as mortality measured after 18 to 20 days
|
Bemisia tabaci
|
0.4
ugAi/ml
|
|
Journal : Crop Protection
Year : 2012
Volume : 40
First Page : 16
Last Page : 21
|
Insecticidal activity against Bemisia tabaci Bupro-SEL (sweet potato whitefly) from 10 generation buprofezin resistance assessed as mortality measured after 18 to 20 days
|
Bemisia tabaci
|
4.04
ugAi/ml
|
|
Journal : Crop Protection
Year : 2012
Volume : 40
First Page : 16
Last Page : 21
|
Insecticidal activity against Bemisia tabaci UNSEL G11 (sweet potato whitefly) from 11 generation buprofezin resistance assessed as mortality measured after 18 to 20 days
|
Bemisia tabaci
|
0.36
ugAi/ml
|
|
Journal : Crop Protection
Year : 2012
Volume : 40
First Page : 16
Last Page : 21
|
Insecticidal activity against Bemisia tabaci (sweet potato whitefly) from one generation buprofezin resistance G1 assessed as mortality measured after 18 to 20 days
|
Bemisia tabaci
|
2.21
ugAi/ml
|
|
Journal : Crop Protection
Year : 2012
Volume : 40
First Page : 16
Last Page : 21
|
Insecticidal activity against Bemisia tabaci (sweet potato whitefly) susceptible Lab-PK assessed as mortality measured after 18 to 20 days
|
Bemisia tabaci
|
0.39
ugAi/ml
|
|
Journal : Crop Protection
Year : 2012
Volume : 40
First Page : 16
Last Page : 21
|
Risk quotient, recommended field rate (g/ha) to LC50 for Trichogramma nubilale (mg/L)
|
Trichogramma nubilale
|
1.17
|
|
Journal : Crop Protection
Title : Susceptibility of adult Trichogramma nubilale (Hymenoptera: Trichogrammatidae) to selected insecticides with different modes of action
Year : 2012
Volume : 34
First Page : 76
Last Page : 72
Authors : Wang Y, Yu R, Zhao X, Chen L, Wu C, Cang T, Wang Q.
Abstract : The parasitic wasp Trichogramma nubilale (Hymenoptera: Trichogrammatidae) is one of the most suitable parasitoids for controlling Asian corn borer, Ostrinia furnacalis (Lepidoptera: Crambidae). Although extensive toxicological tests have been carried out to elucidate the toxicities of insecticides to trichogrammatids, the acute toxicity risks of commonly used insecticides to T. nubilale are not well known. Among the 7 classes of tested chemicals, organophosphates and carbamates had the highest intrinsic toxicity to the parasitoid with LC50 values ranging from 0.081 (0.062–0.12) to 2.10 (1.23–3.47) and from 0.12 (0.11–0.14) to 0.95 (0.87–1.05) mg a.i. per liter, respectively. The phenylpyrazoles (with the exception of butene-fipronil), avermectins, neonicotinoids and pyrethroids induced intermediate toxicity responses with LC50 values ranging from 0.29 to 4.67, 2.36 to 11.27, 1.86 to 311.9, and 10.98–150.3 mg a.i. per liter, respectively. In contrast, insect growth regulators (IGRs) exhibited the least toxicity to the parasitoid with LC50 values ranging from 3452 (3114–3877) to 10,168 (8848–12,027) mg a.i. per liter. A risk quotient analysis indicated that neonicotinoids, avermectins, pyrethroids, IGRs and phenylpyrazoles (with the exception of butene-fipronil) were safe, but organophosphates and carbamates were slightly to moderately toxic or highly toxic to T. nubilale. This study provided informative data for implementing both biological and chemical control strategies in integrated pest management (IPM) of corn lepidopterans.
|
Contact toxicity against Trichogramma nubilale assessed as mortality after 24 hr by modified slight dry film method
|
Trichogramma nubilale
|
167.9
mgAi/L
|
|
Contact toxicity against Trichogramma nubilale assessed as mortality after 24 hr by modified slight dry film method
|
Trichogramma nubilale
|
19.24
mgAi/L
|
|
Journal : Crop Protection
Title : Susceptibility of adult Trichogramma nubilale (Hymenoptera: Trichogrammatidae) to selected insecticides with different modes of action
Year : 2012
Volume : 34
First Page : 76
Last Page : 72
Authors : Wang Y, Yu R, Zhao X, Chen L, Wu C, Cang T, Wang Q.
Abstract : The parasitic wasp Trichogramma nubilale (Hymenoptera: Trichogrammatidae) is one of the most suitable parasitoids for controlling Asian corn borer, Ostrinia furnacalis (Lepidoptera: Crambidae). Although extensive toxicological tests have been carried out to elucidate the toxicities of insecticides to trichogrammatids, the acute toxicity risks of commonly used insecticides to T. nubilale are not well known. Among the 7 classes of tested chemicals, organophosphates and carbamates had the highest intrinsic toxicity to the parasitoid with LC50 values ranging from 0.081 (0.062–0.12) to 2.10 (1.23–3.47) and from 0.12 (0.11–0.14) to 0.95 (0.87–1.05) mg a.i. per liter, respectively. The phenylpyrazoles (with the exception of butene-fipronil), avermectins, neonicotinoids and pyrethroids induced intermediate toxicity responses with LC50 values ranging from 0.29 to 4.67, 2.36 to 11.27, 1.86 to 311.9, and 10.98–150.3 mg a.i. per liter, respectively. In contrast, insect growth regulators (IGRs) exhibited the least toxicity to the parasitoid with LC50 values ranging from 3452 (3114–3877) to 10,168 (8848–12,027) mg a.i. per liter. A risk quotient analysis indicated that neonicotinoids, avermectins, pyrethroids, IGRs and phenylpyrazoles (with the exception of butene-fipronil) were safe, but organophosphates and carbamates were slightly to moderately toxic or highly toxic to T. nubilale. This study provided informative data for implementing both biological and chemical control strategies in integrated pest management (IPM) of corn lepidopterans.
|
Binding affinity to freshwater snail Lymnaea stagnalis AChBP assessed as [3H]EPI binding by radioligand binding assay
|
Lymnaea stagnalis
|
1200.0
nM
|
|
Journal : J Agric Food Chem
Title : Unique neonicotinoid binding conformations conferring selective receptor interactions.
Year : 2011
Volume : 59
Issue : 7
First Page : 2825
Last Page : 2828
Authors : Tomizawa M, Casida JE.
Abstract : Neonicotinoid agonists selectively act on the insect nicotinic acetylcholine receptor (nAChR). The molecular basis for this specificity is deciphered by comparisons of two acetylcholine binding proteins (AChBPs) with distinct pharmacological profiles that serve as structural homologues for the nAChR subtypes. Aplysia AChBP has high neonicotinoid sensitivity, whereas Lymnaea AChBP has low neonicotinoid sensitivity, pharmacologies reminiscent of insect and vertebrate nAChR subtypes, respectively. The ligand-receptor interactions for these AChBPs were established by chemical and structural neurobiology approaches. Neonicotinoids and nicotinoids bind in a single conformation with Aplysia AChBP, wherein the electronegative nitro or cyano pharmacophore of the neonicotinoid faces in a reversed orientation relative to the cationic nicotinoid functionality. For Lymnaea AChBP, the neonicotinoids have two binding conformations in this vertebrate receptor model, which are completely inverted relative to each other, whereas nicotinoids are nestled in only one conserved conformation. Therefore, the unique binding conformations of nicotinic agonists determine the selective receptor interactions.
|
Binding affinity to salt water mollusc Aplysia californica AChBP Y55W mutant assessed as [3H]acetamiprid binding by radioligand binding assay
|
Aplysia californica
|
32.0
nM
|
|
Journal : J Agric Food Chem
Title : Unique neonicotinoid binding conformations conferring selective receptor interactions.
Year : 2011
Volume : 59
Issue : 7
First Page : 2825
Last Page : 2828
Authors : Tomizawa M, Casida JE.
Abstract : Neonicotinoid agonists selectively act on the insect nicotinic acetylcholine receptor (nAChR). The molecular basis for this specificity is deciphered by comparisons of two acetylcholine binding proteins (AChBPs) with distinct pharmacological profiles that serve as structural homologues for the nAChR subtypes. Aplysia AChBP has high neonicotinoid sensitivity, whereas Lymnaea AChBP has low neonicotinoid sensitivity, pharmacologies reminiscent of insect and vertebrate nAChR subtypes, respectively. The ligand-receptor interactions for these AChBPs were established by chemical and structural neurobiology approaches. Neonicotinoids and nicotinoids bind in a single conformation with Aplysia AChBP, wherein the electronegative nitro or cyano pharmacophore of the neonicotinoid faces in a reversed orientation relative to the cationic nicotinoid functionality. For Lymnaea AChBP, the neonicotinoids have two binding conformations in this vertebrate receptor model, which are completely inverted relative to each other, whereas nicotinoids are nestled in only one conserved conformation. Therefore, the unique binding conformations of nicotinic agonists determine the selective receptor interactions.
|
Binding affinity to recombinant Gallus gallus (chicken) alpha4beta2 nicotinic acetylcholine receptor assessed as [3H]NIC binding by radioligand binding assay
|
Gallus gallus
|
680.0
nM
|
|
Journal : J Agric Food Chem
Title : Unique neonicotinoid binding conformations conferring selective receptor interactions.
Year : 2011
Volume : 59
Issue : 7
First Page : 2825
Last Page : 2828
Authors : Tomizawa M, Casida JE.
Abstract : Neonicotinoid agonists selectively act on the insect nicotinic acetylcholine receptor (nAChR). The molecular basis for this specificity is deciphered by comparisons of two acetylcholine binding proteins (AChBPs) with distinct pharmacological profiles that serve as structural homologues for the nAChR subtypes. Aplysia AChBP has high neonicotinoid sensitivity, whereas Lymnaea AChBP has low neonicotinoid sensitivity, pharmacologies reminiscent of insect and vertebrate nAChR subtypes, respectively. The ligand-receptor interactions for these AChBPs were established by chemical and structural neurobiology approaches. Neonicotinoids and nicotinoids bind in a single conformation with Aplysia AChBP, wherein the electronegative nitro or cyano pharmacophore of the neonicotinoid faces in a reversed orientation relative to the cationic nicotinoid functionality. For Lymnaea AChBP, the neonicotinoids have two binding conformations in this vertebrate receptor model, which are completely inverted relative to each other, whereas nicotinoids are nestled in only one conserved conformation. Therefore, the unique binding conformations of nicotinic agonists determine the selective receptor interactions.
|
Binding affinity to Drosophila brain nicotinic acetylcholine receptor assessed as [3H]IMI binding by radioligand binding assay
|
Drosophila
|
7.2
nM
|
|
Journal : J Agric Food Chem
Title : Unique neonicotinoid binding conformations conferring selective receptor interactions.
Year : 2011
Volume : 59
Issue : 7
First Page : 2825
Last Page : 2828
Authors : Tomizawa M, Casida JE.
Abstract : Neonicotinoid agonists selectively act on the insect nicotinic acetylcholine receptor (nAChR). The molecular basis for this specificity is deciphered by comparisons of two acetylcholine binding proteins (AChBPs) with distinct pharmacological profiles that serve as structural homologues for the nAChR subtypes. Aplysia AChBP has high neonicotinoid sensitivity, whereas Lymnaea AChBP has low neonicotinoid sensitivity, pharmacologies reminiscent of insect and vertebrate nAChR subtypes, respectively. The ligand-receptor interactions for these AChBPs were established by chemical and structural neurobiology approaches. Neonicotinoids and nicotinoids bind in a single conformation with Aplysia AChBP, wherein the electronegative nitro or cyano pharmacophore of the neonicotinoid faces in a reversed orientation relative to the cationic nicotinoid functionality. For Lymnaea AChBP, the neonicotinoids have two binding conformations in this vertebrate receptor model, which are completely inverted relative to each other, whereas nicotinoids are nestled in only one conserved conformation. Therefore, the unique binding conformations of nicotinic agonists determine the selective receptor interactions.
|
Ratio of LC50 for third-instar Apolygus lucorum reared in asparagus bean pod at 15 degC to LC50 for third-instar Apolygus lucorum reared in asparagus bean pod at 25 degC
|
Apolygus lucorum
|
3.83
|
|
Journal : J Pesticide Sci
Year : 2012
Volume : 37
Issue : 2
First Page : 135
Last Page : 139
|
Ratio of LC50 for third-instar Apolygus lucorum reared in asparagus bean pod at 15 degC to LC50 for third-instar Apolygus lucorum reared in asparagus bean pod at 35 degC
|
Apolygus lucorum
|
8.87
|
|
Journal : J Pesticide Sci
Year : 2012
Volume : 37
Issue : 2
First Page : 135
Last Page : 139
|
Ratio of LC50 for third-instar Apolygus lucorum reared in asparagus bean pod at 15 degC to LC50 for third-instar Apolygus lucorum reared in asparagus bean pod at 30 degC
|
Apolygus lucorum
|
6.11
|
|
Journal : J Pesticide Sci
Year : 2012
Volume : 37
Issue : 2
First Page : 135
Last Page : 139
|
Ratio of LC50 for third-instar Apolygus lucorum reared in asparagus bean pod at 15 degC to LC50 for third-instar Apolygus lucorum reared in asparagus bean pod at 20 degC
|
Apolygus lucorum
|
1.66
|
|
Journal : J Pesticide Sci
Year : 2012
Volume : 37
Issue : 2
First Page : 135
Last Page : 139
|
Insecticidal activity against third-instar Apolygus lucorum reared in asparagus bean pod assessed as mortality administered by dipping pods in compound solution measured after 48 hr at 35 degC
|
Apolygus lucorum
|
181.95
ug.mL-1
|
|
Journal : J Pesticide Sci
Year : 2012
Volume : 37
Issue : 2
First Page : 135
Last Page : 139
|
Insecticidal activity against third-instar Apolygus lucorum reared in asparagus bean pod assessed as mortality administered by dipping pods in compound solution measured after 48 hr at 30 degC
|
Apolygus lucorum
|
264.02
ug.mL-1
|
|
Journal : J Pesticide Sci
Year : 2012
Volume : 37
Issue : 2
First Page : 135
Last Page : 139
|
Insecticidal activity against third-instar Apolygus lucorum reared in asparagus bean pod assessed as mortality administered by dipping pods in compound solution measured after 48 hr at 25 degC
|
Apolygus lucorum
|
420.95
ug.mL-1
|
|
Journal : J Pesticide Sci
Year : 2012
Volume : 37
Issue : 2
First Page : 135
Last Page : 139
|
Insecticidal activity against third-instar Apolygus lucorum reared in asparagus bean pod assessed as mortality administered by dipping pods in compound solution measured after 48 hr at 20 degC
|
Apolygus lucorum
|
972.05
ug.mL-1
|
|
Journal : J Pesticide Sci
Year : 2012
Volume : 37
Issue : 2
First Page : 135
Last Page : 139
|
Insecticidal activity against third-instar Apolygus lucorum reared in asparagus bean pod assessed as mortality administered by dipping pods in compound solution measured after 48 hr at 15 degC
|
Apolygus lucorum
|
1613.76
ug.mL-1
|
|
Journal : J Pesticide Sci
Year : 2012
Volume : 37
Issue : 2
First Page : 135
Last Page : 139
|
Displacement of [3H]IMI from nicotinic acetylcholine receptor in Drosophila melanogaster head membrane after 90 min by filter binding assay
|
Drosophila melanogaster
|
8.3
nM
|
|
Journal : J. Neurochem.
Year : 2000
Volume : 75
Issue : 3
First Page : 1294
Last Page : 1303
|
Displacement of [3H]IMI from nicotinic acetylcholine receptor in Myzus persicae (green peach aphid) whole body membrane after 90 min by filter binding assay
|
Myzus persicae
|
10.0
nM
|
|
Journal : J. Neurochem.
Year : 2000
Volume : 75
Issue : 3
First Page : 1294
Last Page : 1303
|
Ratio of EC50 for Daphnia magna to EC50 for first instar larvae of Cheumatopsyche brevilineata
|
None
|
4.17
|
|
Journal : J Pesticide Sci
Title : A useful new insecticide bioassay using first-instar larvae of a net-spinning caddisfly, Cheumatopsyche brevilineata (Trichoptera: Hydropsychidae)
Year : 2009
Volume : 34
Issue : 1
First Page : 13
Last Page : 20
Authors : Yokoyama A, Ohtsu K, Iwafune T, Nagai T, Ishihara S, Kobara Y, Horio T, Endo S
|
Insecticidal activity against first-instar larvae of Cheumatopsyche brevilineata after 48 hr
|
Cheumatopsyche
|
0.00335
ug.mL-1
|
|
Journal : J Pesticide Sci
Title : A useful new insecticide bioassay using first-instar larvae of a net-spinning caddisfly, Cheumatopsyche brevilineata (Trichoptera: Hydropsychidae)
Year : 2009
Volume : 34
Issue : 1
First Page : 13
Last Page : 20
Authors : Yokoyama A, Ohtsu K, Iwafune T, Nagai T, Ishihara S, Kobara Y, Horio T, Endo S
|
Octanol-water partition coefficient, log KOW of the compound
|
None
|
0.8
|
|
Journal : J Pesticide Sci
Title : A useful new insecticide bioassay using first-instar larvae of a net-spinning caddisfly, Cheumatopsyche brevilineata (Trichoptera: Hydropsychidae)
Year : 2009
Volume : 34
Issue : 1
First Page : 13
Last Page : 20
Authors : Yokoyama A, Ohtsu K, Iwafune T, Nagai T, Ishihara S, Kobara Y, Horio T, Endo S
|
Insecticidal activity against Daphnia magna after 48 hr
|
Daphnia magna
|
50.0
ug.mL-1
|
|
Journal : J Pesticide Sci
Title : A useful new insecticide bioassay using first-instar larvae of a net-spinning caddisfly, Cheumatopsyche brevilineata (Trichoptera: Hydropsychidae)
Year : 2009
Volume : 34
Issue : 1
First Page : 13
Last Page : 20
Authors : Yokoyama A, Ohtsu K, Iwafune T, Nagai T, Ishihara S, Kobara Y, Horio T, Endo S
|
Resistance ratio of LC50 for Bemisia tabaci (sweet potato whitefly) infested poinsettia leaves to LC50 for Bemisia tabaci (sweet potato whitefly) infested cucumber leaves
|
Bemisia tabaci
|
10.34
|
|
Journal : Pest Manag Sci
Title : Induction effects of host plants on insecticide susceptibility and detoxification enzymes of Bemisia tabaci (Hemiptera: Aleyrodidae).
Year : 2011
Volume : 67
Issue : 1
First Page : 87
Last Page : 93
Authors : Xie W, Wang S, Wu Q, Feng Y, Pan H, Jiao X, Zhou L, Yang X, Fu W, Teng H, Xu B, Zhang Y.
Abstract : BACKGROUND: The polyphagous B-biotype Bemisia tabaci (Gennadius) has developed a high resistance to commonly used insecticides in China. To illustrate the induced changes by host plant, bioassay and biochemical research on five different host populations were investigated. RESULTS: Except for bifenthrin, all tested insecticides showed lower toxicity to the B. tabaci poinsettia population compared with other host populations. Moreover, four insecticides, the exceptions being abamectin and fipronil, showed highest toxicity towards the tomato population. The LC(50) values of the poinsettia population, particularly towards acetamiprid, were 14.8-, 10.3- and 7.29-fold higher than those of tomato, cucumber and cabbage respectively. The CarE activities of B. tabaci cabbage and cucumber populations were all significantly higher than those of poinsettia, cotton and tomato populations. The ratio of the cabbage population was 1.97-, 1.79- and 1.30-fold higher than that of poinsettia, cotton and tomato respectively. The frequency profiles for this activity also have obvious differences. The GST and P450 activities of the cucumber population were the lowest in the five host populations. CONCLUSION: Long-term induction of host plants for B-biotype B. tabaci could influence their susceptibilities to several insecticides. Rational selection and usage of insecticides for particular hosts will be helpful for resistance management and control of this species.
|
Insecticidal activity against Bemisia tabaci (sweet potato whitefly) infested cabbage leaves assessed as mortality after 48 hr by leaf dip method
|
Bemisia tabaci
|
17.7
ug.mL-1
|
|
Journal : Pest Manag Sci
Title : Induction effects of host plants on insecticide susceptibility and detoxification enzymes of Bemisia tabaci (Hemiptera: Aleyrodidae).
Year : 2011
Volume : 67
Issue : 1
First Page : 87
Last Page : 93
Authors : Xie W, Wang S, Wu Q, Feng Y, Pan H, Jiao X, Zhou L, Yang X, Fu W, Teng H, Xu B, Zhang Y.
Abstract : BACKGROUND: The polyphagous B-biotype Bemisia tabaci (Gennadius) has developed a high resistance to commonly used insecticides in China. To illustrate the induced changes by host plant, bioassay and biochemical research on five different host populations were investigated. RESULTS: Except for bifenthrin, all tested insecticides showed lower toxicity to the B. tabaci poinsettia population compared with other host populations. Moreover, four insecticides, the exceptions being abamectin and fipronil, showed highest toxicity towards the tomato population. The LC(50) values of the poinsettia population, particularly towards acetamiprid, were 14.8-, 10.3- and 7.29-fold higher than those of tomato, cucumber and cabbage respectively. The CarE activities of B. tabaci cabbage and cucumber populations were all significantly higher than those of poinsettia, cotton and tomato populations. The ratio of the cabbage population was 1.97-, 1.79- and 1.30-fold higher than that of poinsettia, cotton and tomato respectively. The frequency profiles for this activity also have obvious differences. The GST and P450 activities of the cucumber population were the lowest in the five host populations. CONCLUSION: Long-term induction of host plants for B-biotype B. tabaci could influence their susceptibilities to several insecticides. Rational selection and usage of insecticides for particular hosts will be helpful for resistance management and control of this species.
|
Resistance ratio of LC50 for Bemisia tabaci (sweet potato whitefly) infested poinsettia leaves to LC50 for Bemisia tabaci (sweet potato whitefly) infested tomato leaves
|
Bemisia tabaci
|
14.8
|
|
Journal : Pest Manag Sci
Title : Induction effects of host plants on insecticide susceptibility and detoxification enzymes of Bemisia tabaci (Hemiptera: Aleyrodidae).
Year : 2011
Volume : 67
Issue : 1
First Page : 87
Last Page : 93
Authors : Xie W, Wang S, Wu Q, Feng Y, Pan H, Jiao X, Zhou L, Yang X, Fu W, Teng H, Xu B, Zhang Y.
Abstract : BACKGROUND: The polyphagous B-biotype Bemisia tabaci (Gennadius) has developed a high resistance to commonly used insecticides in China. To illustrate the induced changes by host plant, bioassay and biochemical research on five different host populations were investigated. RESULTS: Except for bifenthrin, all tested insecticides showed lower toxicity to the B. tabaci poinsettia population compared with other host populations. Moreover, four insecticides, the exceptions being abamectin and fipronil, showed highest toxicity towards the tomato population. The LC(50) values of the poinsettia population, particularly towards acetamiprid, were 14.8-, 10.3- and 7.29-fold higher than those of tomato, cucumber and cabbage respectively. The CarE activities of B. tabaci cabbage and cucumber populations were all significantly higher than those of poinsettia, cotton and tomato populations. The ratio of the cabbage population was 1.97-, 1.79- and 1.30-fold higher than that of poinsettia, cotton and tomato respectively. The frequency profiles for this activity also have obvious differences. The GST and P450 activities of the cucumber population were the lowest in the five host populations. CONCLUSION: Long-term induction of host plants for B-biotype B. tabaci could influence their susceptibilities to several insecticides. Rational selection and usage of insecticides for particular hosts will be helpful for resistance management and control of this species.
|
Insecticidal activity against Bemisia tabaci (sweet potato whitefly) infested tomato leaves assessed as mortality after 48 hr by leaf dip method
|
Bemisia tabaci
|
8.73
ug.mL-1
|
|
Journal : Pest Manag Sci
Title : Induction effects of host plants on insecticide susceptibility and detoxification enzymes of Bemisia tabaci (Hemiptera: Aleyrodidae).
Year : 2011
Volume : 67
Issue : 1
First Page : 87
Last Page : 93
Authors : Xie W, Wang S, Wu Q, Feng Y, Pan H, Jiao X, Zhou L, Yang X, Fu W, Teng H, Xu B, Zhang Y.
Abstract : BACKGROUND: The polyphagous B-biotype Bemisia tabaci (Gennadius) has developed a high resistance to commonly used insecticides in China. To illustrate the induced changes by host plant, bioassay and biochemical research on five different host populations were investigated. RESULTS: Except for bifenthrin, all tested insecticides showed lower toxicity to the B. tabaci poinsettia population compared with other host populations. Moreover, four insecticides, the exceptions being abamectin and fipronil, showed highest toxicity towards the tomato population. The LC(50) values of the poinsettia population, particularly towards acetamiprid, were 14.8-, 10.3- and 7.29-fold higher than those of tomato, cucumber and cabbage respectively. The CarE activities of B. tabaci cabbage and cucumber populations were all significantly higher than those of poinsettia, cotton and tomato populations. The ratio of the cabbage population was 1.97-, 1.79- and 1.30-fold higher than that of poinsettia, cotton and tomato respectively. The frequency profiles for this activity also have obvious differences. The GST and P450 activities of the cucumber population were the lowest in the five host populations. CONCLUSION: Long-term induction of host plants for B-biotype B. tabaci could influence their susceptibilities to several insecticides. Rational selection and usage of insecticides for particular hosts will be helpful for resistance management and control of this species.
|
Insecticidal activity against Bemisia tabaci (sweet potato whitefly) infested cucumber leaves assessed as mortality after 48 hr by leaf dip method
|
Bemisia tabaci
|
12.4
ug.mL-1
|
|
Journal : Pest Manag Sci
Title : Induction effects of host plants on insecticide susceptibility and detoxification enzymes of Bemisia tabaci (Hemiptera: Aleyrodidae).
Year : 2011
Volume : 67
Issue : 1
First Page : 87
Last Page : 93
Authors : Xie W, Wang S, Wu Q, Feng Y, Pan H, Jiao X, Zhou L, Yang X, Fu W, Teng H, Xu B, Zhang Y.
Abstract : BACKGROUND: The polyphagous B-biotype Bemisia tabaci (Gennadius) has developed a high resistance to commonly used insecticides in China. To illustrate the induced changes by host plant, bioassay and biochemical research on five different host populations were investigated. RESULTS: Except for bifenthrin, all tested insecticides showed lower toxicity to the B. tabaci poinsettia population compared with other host populations. Moreover, four insecticides, the exceptions being abamectin and fipronil, showed highest toxicity towards the tomato population. The LC(50) values of the poinsettia population, particularly towards acetamiprid, were 14.8-, 10.3- and 7.29-fold higher than those of tomato, cucumber and cabbage respectively. The CarE activities of B. tabaci cabbage and cucumber populations were all significantly higher than those of poinsettia, cotton and tomato populations. The ratio of the cabbage population was 1.97-, 1.79- and 1.30-fold higher than that of poinsettia, cotton and tomato respectively. The frequency profiles for this activity also have obvious differences. The GST and P450 activities of the cucumber population were the lowest in the five host populations. CONCLUSION: Long-term induction of host plants for B-biotype B. tabaci could influence their susceptibilities to several insecticides. Rational selection and usage of insecticides for particular hosts will be helpful for resistance management and control of this species.
|
Insecticidal activity against Bemisia tabaci (sweet potato whitefly) infested cotton leaves assessed as mortality after 48 hr by leaf dip method
|
Bemisia tabaci
|
66.4
ug.mL-1
|
|
Journal : Pest Manag Sci
Title : Induction effects of host plants on insecticide susceptibility and detoxification enzymes of Bemisia tabaci (Hemiptera: Aleyrodidae).
Year : 2011
Volume : 67
Issue : 1
First Page : 87
Last Page : 93
Authors : Xie W, Wang S, Wu Q, Feng Y, Pan H, Jiao X, Zhou L, Yang X, Fu W, Teng H, Xu B, Zhang Y.
Abstract : BACKGROUND: The polyphagous B-biotype Bemisia tabaci (Gennadius) has developed a high resistance to commonly used insecticides in China. To illustrate the induced changes by host plant, bioassay and biochemical research on five different host populations were investigated. RESULTS: Except for bifenthrin, all tested insecticides showed lower toxicity to the B. tabaci poinsettia population compared with other host populations. Moreover, four insecticides, the exceptions being abamectin and fipronil, showed highest toxicity towards the tomato population. The LC(50) values of the poinsettia population, particularly towards acetamiprid, were 14.8-, 10.3- and 7.29-fold higher than those of tomato, cucumber and cabbage respectively. The CarE activities of B. tabaci cabbage and cucumber populations were all significantly higher than those of poinsettia, cotton and tomato populations. The ratio of the cabbage population was 1.97-, 1.79- and 1.30-fold higher than that of poinsettia, cotton and tomato respectively. The frequency profiles for this activity also have obvious differences. The GST and P450 activities of the cucumber population were the lowest in the five host populations. CONCLUSION: Long-term induction of host plants for B-biotype B. tabaci could influence their susceptibilities to several insecticides. Rational selection and usage of insecticides for particular hosts will be helpful for resistance management and control of this species.
|
Insecticidal activity against Bemisia tabaci (sweet potato whitefly) infested poinsettia leaves assessed as mortality after 48 hr by leaf dip method
|
Bemisia tabaci
|
129.0
ug.mL-1
|
|
Journal : Pest Manag Sci
Title : Induction effects of host plants on insecticide susceptibility and detoxification enzymes of Bemisia tabaci (Hemiptera: Aleyrodidae).
Year : 2011
Volume : 67
Issue : 1
First Page : 87
Last Page : 93
Authors : Xie W, Wang S, Wu Q, Feng Y, Pan H, Jiao X, Zhou L, Yang X, Fu W, Teng H, Xu B, Zhang Y.
Abstract : BACKGROUND: The polyphagous B-biotype Bemisia tabaci (Gennadius) has developed a high resistance to commonly used insecticides in China. To illustrate the induced changes by host plant, bioassay and biochemical research on five different host populations were investigated. RESULTS: Except for bifenthrin, all tested insecticides showed lower toxicity to the B. tabaci poinsettia population compared with other host populations. Moreover, four insecticides, the exceptions being abamectin and fipronil, showed highest toxicity towards the tomato population. The LC(50) values of the poinsettia population, particularly towards acetamiprid, were 14.8-, 10.3- and 7.29-fold higher than those of tomato, cucumber and cabbage respectively. The CarE activities of B. tabaci cabbage and cucumber populations were all significantly higher than those of poinsettia, cotton and tomato populations. The ratio of the cabbage population was 1.97-, 1.79- and 1.30-fold higher than that of poinsettia, cotton and tomato respectively. The frequency profiles for this activity also have obvious differences. The GST and P450 activities of the cucumber population were the lowest in the five host populations. CONCLUSION: Long-term induction of host plants for B-biotype B. tabaci could influence their susceptibilities to several insecticides. Rational selection and usage of insecticides for particular hosts will be helpful for resistance management and control of this species.
|
Resistance factor, ratio of LC50 for adult female Bemisia tabaci B-biotype NJ-Imi (sweet potato whitefly) (G30) to LC50 for adult female B-type Bemisia tabaci NJ
|
Bemisia tabaci
|
143.0
|
|
Journal : Pest Manag Sci
Title : Cross-resistance, inheritance and biochemical mechanisms of imidacloprid resistance in B-biotype Bemisia tabaci.
Year : 2009
Volume : 65
Issue : 11
First Page : 1189
Last Page : 1194
Authors : Wang Z, Yao M, Wu Y.
Abstract : BACKGROUND: The B-type Bemisia tabaci (Gennadius) has become established in many regions in China, and neonicotinoids are extensively used to control this pest. Imidacloprid resistance in a laboratory-selected strain of B-type B. tabaci was characterised in order to provide the basis for recommending resistance management tactics. RESULTS: The NJ-Imi strain of B-type B. tabaci was selected from the NJ strain with imidacloprid for 30 generations. The NJ-Imi strain exhibited 490-fold resistance to imidacloprid, high levels of cross-resistance to three other neonicotinoids, low levels of cross-resistance to monosultap, cartap and spinosad, but no cross-resistance to abamectin and cypermethrin. Imidacloprid resistance in the NJ-Imi strain was autosomal and semi-dominant. It is shown that enhanced detoxification mediated by cytochrome-P450-dependent monooxygenases contributes to imidacloprid resistance to some extent in the NJ-Imi strain. Results from synergist bioassays and cross-resistance patterns indicated that target-site insensitivity may be involved in imidacloprid resistance in the NJ-Imi strain of B. tabaci. CONCLUSION: Although oxidative detoxification mediated by P450 monooxygenases is involved in imidacloprid resistance in the NJ-Imi strain of B-type B. tabaci, target-site modification as an additional resistance mechanism cannot be ruled out. Considering the high risk of cross-resistance, neonicotinoids should be regarded as a single group when implementing an insecticide rotation scheme in B. tabaci control.
|
Resistance factor, ratio of LC50 for adult female Bemisia tabaci B-biotype NJ-Imi (sweet potato whitefly) (G15) to LC50 for adult female B-type Bemisia tabaci NJ
|
Bemisia tabaci
|
49.0
|
|
Journal : Pest Manag Sci
Title : Cross-resistance, inheritance and biochemical mechanisms of imidacloprid resistance in B-biotype Bemisia tabaci.
Year : 2009
Volume : 65
Issue : 11
First Page : 1189
Last Page : 1194
Authors : Wang Z, Yao M, Wu Y.
Abstract : BACKGROUND: The B-type Bemisia tabaci (Gennadius) has become established in many regions in China, and neonicotinoids are extensively used to control this pest. Imidacloprid resistance in a laboratory-selected strain of B-type B. tabaci was characterised in order to provide the basis for recommending resistance management tactics. RESULTS: The NJ-Imi strain of B-type B. tabaci was selected from the NJ strain with imidacloprid for 30 generations. The NJ-Imi strain exhibited 490-fold resistance to imidacloprid, high levels of cross-resistance to three other neonicotinoids, low levels of cross-resistance to monosultap, cartap and spinosad, but no cross-resistance to abamectin and cypermethrin. Imidacloprid resistance in the NJ-Imi strain was autosomal and semi-dominant. It is shown that enhanced detoxification mediated by cytochrome-P450-dependent monooxygenases contributes to imidacloprid resistance to some extent in the NJ-Imi strain. Results from synergist bioassays and cross-resistance patterns indicated that target-site insensitivity may be involved in imidacloprid resistance in the NJ-Imi strain of B. tabaci. CONCLUSION: Although oxidative detoxification mediated by P450 monooxygenases is involved in imidacloprid resistance in the NJ-Imi strain of B-type B. tabaci, target-site modification as an additional resistance mechanism cannot be ruled out. Considering the high risk of cross-resistance, neonicotinoids should be regarded as a single group when implementing an insecticide rotation scheme in B. tabaci control.
|
Insecticidal activity against adult female imidacloprid-resistant Bemisia tabaci B-biotype NJ-Imi (sweet potato whitefly) (G30) in cotton leaf discs assessed as mortality treated for 10 secs prior to adult infestation measured after 72 hr by leaf-dip bioassay
|
Bemisia tabaci
|
186.0
ug.mL-1
|
|
Journal : Pest Manag Sci
Title : Cross-resistance, inheritance and biochemical mechanisms of imidacloprid resistance in B-biotype Bemisia tabaci.
Year : 2009
Volume : 65
Issue : 11
First Page : 1189
Last Page : 1194
Authors : Wang Z, Yao M, Wu Y.
Abstract : BACKGROUND: The B-type Bemisia tabaci (Gennadius) has become established in many regions in China, and neonicotinoids are extensively used to control this pest. Imidacloprid resistance in a laboratory-selected strain of B-type B. tabaci was characterised in order to provide the basis for recommending resistance management tactics. RESULTS: The NJ-Imi strain of B-type B. tabaci was selected from the NJ strain with imidacloprid for 30 generations. The NJ-Imi strain exhibited 490-fold resistance to imidacloprid, high levels of cross-resistance to three other neonicotinoids, low levels of cross-resistance to monosultap, cartap and spinosad, but no cross-resistance to abamectin and cypermethrin. Imidacloprid resistance in the NJ-Imi strain was autosomal and semi-dominant. It is shown that enhanced detoxification mediated by cytochrome-P450-dependent monooxygenases contributes to imidacloprid resistance to some extent in the NJ-Imi strain. Results from synergist bioassays and cross-resistance patterns indicated that target-site insensitivity may be involved in imidacloprid resistance in the NJ-Imi strain of B. tabaci. CONCLUSION: Although oxidative detoxification mediated by P450 monooxygenases is involved in imidacloprid resistance in the NJ-Imi strain of B-type B. tabaci, target-site modification as an additional resistance mechanism cannot be ruled out. Considering the high risk of cross-resistance, neonicotinoids should be regarded as a single group when implementing an insecticide rotation scheme in B. tabaci control.
|
Insecticidal activity against adult female imidacloprid-resistant Bemisia tabaci B-biotype NJ-Imi (sweet potato whitefly) (G15) in cotton leaf discs assessed as mortality treated for 10 secs prior to adult infestation measured after 72 hr by leaf-dip bioassay
|
Bemisia tabaci
|
63.0
ug.mL-1
|
|
Journal : Pest Manag Sci
Title : Cross-resistance, inheritance and biochemical mechanisms of imidacloprid resistance in B-biotype Bemisia tabaci.
Year : 2009
Volume : 65
Issue : 11
First Page : 1189
Last Page : 1194
Authors : Wang Z, Yao M, Wu Y.
Abstract : BACKGROUND: The B-type Bemisia tabaci (Gennadius) has become established in many regions in China, and neonicotinoids are extensively used to control this pest. Imidacloprid resistance in a laboratory-selected strain of B-type B. tabaci was characterised in order to provide the basis for recommending resistance management tactics. RESULTS: The NJ-Imi strain of B-type B. tabaci was selected from the NJ strain with imidacloprid for 30 generations. The NJ-Imi strain exhibited 490-fold resistance to imidacloprid, high levels of cross-resistance to three other neonicotinoids, low levels of cross-resistance to monosultap, cartap and spinosad, but no cross-resistance to abamectin and cypermethrin. Imidacloprid resistance in the NJ-Imi strain was autosomal and semi-dominant. It is shown that enhanced detoxification mediated by cytochrome-P450-dependent monooxygenases contributes to imidacloprid resistance to some extent in the NJ-Imi strain. Results from synergist bioassays and cross-resistance patterns indicated that target-site insensitivity may be involved in imidacloprid resistance in the NJ-Imi strain of B. tabaci. CONCLUSION: Although oxidative detoxification mediated by P450 monooxygenases is involved in imidacloprid resistance in the NJ-Imi strain of B-type B. tabaci, target-site modification as an additional resistance mechanism cannot be ruled out. Considering the high risk of cross-resistance, neonicotinoids should be regarded as a single group when implementing an insecticide rotation scheme in B. tabaci control.
|
Insecticidal activity against adult female imidacloprid-susceptible Bemisia tabaci B-biotype NJ (sweet potato whitefly) in cotton leaf discs assessed as mortality treated for 10 secs prior to adult infestation measured after 72 hr by leaf-dip bioassay
|
Bemisia tabaci
|
1.3
ug.mL-1
|
|
Journal : Pest Manag Sci
Title : Cross-resistance, inheritance and biochemical mechanisms of imidacloprid resistance in B-biotype Bemisia tabaci.
Year : 2009
Volume : 65
Issue : 11
First Page : 1189
Last Page : 1194
Authors : Wang Z, Yao M, Wu Y.
Abstract : BACKGROUND: The B-type Bemisia tabaci (Gennadius) has become established in many regions in China, and neonicotinoids are extensively used to control this pest. Imidacloprid resistance in a laboratory-selected strain of B-type B. tabaci was characterised in order to provide the basis for recommending resistance management tactics. RESULTS: The NJ-Imi strain of B-type B. tabaci was selected from the NJ strain with imidacloprid for 30 generations. The NJ-Imi strain exhibited 490-fold resistance to imidacloprid, high levels of cross-resistance to three other neonicotinoids, low levels of cross-resistance to monosultap, cartap and spinosad, but no cross-resistance to abamectin and cypermethrin. Imidacloprid resistance in the NJ-Imi strain was autosomal and semi-dominant. It is shown that enhanced detoxification mediated by cytochrome-P450-dependent monooxygenases contributes to imidacloprid resistance to some extent in the NJ-Imi strain. Results from synergist bioassays and cross-resistance patterns indicated that target-site insensitivity may be involved in imidacloprid resistance in the NJ-Imi strain of B. tabaci. CONCLUSION: Although oxidative detoxification mediated by P450 monooxygenases is involved in imidacloprid resistance in the NJ-Imi strain of B-type B. tabaci, target-site modification as an additional resistance mechanism cannot be ruled out. Considering the high risk of cross-resistance, neonicotinoids should be regarded as a single group when implementing an insecticide rotation scheme in B. tabaci control.
|
Toxicity against Metaseiulus occidentalis adults in bean leaf disk infested with two-spotted spider mites assessed as repellency at 0.1167 g AI/L treated using worst-case laboratory exposure procedure measured after 72 hr (Rvb = 16.1%)
|
Metaseiulus occidentalis
|
50.0
%
|
|
Journal : Pest Manag Sci
Year : 2009
Volume : 65
Issue : 6
First Page : 635
Last Page : 639
|
Toxicity against Metaseiulus occidentalis adults in bean leaf disk infested with two-spotted spider mites assessed as repellency at 0.1167 g AI/L treated using worst-case laboratory exposure procedure measured after 48 hr (Rvb = 13.6%)
|
Metaseiulus occidentalis
|
41.0
%
|
|
Journal : Pest Manag Sci
Year : 2009
Volume : 65
Issue : 6
First Page : 635
Last Page : 639
|
Toxicity against Metaseiulus occidentalis adults in bean leaf disk infested with two-spotted spider mites assessed as repellency at 0.1167 g AI/L treated using worst-case laboratory exposure procedure measured after 24 hr (Rvb = 3.6%)
|
Metaseiulus occidentalis
|
16.0
%
|
|
Journal : Pest Manag Sci
Year : 2009
Volume : 65
Issue : 6
First Page : 635
Last Page : 639
|
Toxicity against Metaseiulus occidentalis adults in bean leaf disk infested with two-spotted spider mites assessed as average number of eggs laid (fecundity)at 0.1167 g AI/L treated using worst-case laboratory exposure procedure measured after 72 hr (Rvb = 2.23/day)
|
Metaseiulus occidentalis
|
0.0
/day
|
|
Journal : Pest Manag Sci
Year : 2009
Volume : 65
Issue : 6
First Page : 635
Last Page : 639
|
Toxicity against Metaseiulus occidentalis adults in bean leaf disk infested with two-spotted spider mites assessed as average number of eggs laid (fecundity) at 0.1167 g AI/L treated using worst-case laboratory exposure procedure measured after 48 hr (Rvb = 2.30/day)
|
Metaseiulus occidentalis
|
0.13
/day
|
|
Journal : Pest Manag Sci
Year : 2009
Volume : 65
Issue : 6
First Page : 635
Last Page : 639
|
Toxicity against Metaseiulus occidentalis adults in bean leaf disk infested with two-spotted spider mites assessed as average number of eggs laid (fecundity) at 0.1167 g AI/L treated using worst-case laboratory exposure procedure measured after 24 hr (Rvb = 2.11/day)
|
Metaseiulus occidentalis
|
1.3
/day
|
|
Journal : Pest Manag Sci
Year : 2009
Volume : 65
Issue : 6
First Page : 635
Last Page : 639
|
Toxicity against Metaseiulus occidentalis adults in bean leaf disk infested with two-spotted spider mites assessed as corrected mortality at 0.1167/128 g AI/L treated using worst-case laboratory exposure procedure measured after 72 hr
|
Metaseiulus occidentalis
|
2.4
%
|
|
Journal : Pest Manag Sci
Year : 2009
Volume : 65
Issue : 6
First Page : 635
Last Page : 639
|
Toxicity against Metaseiulus occidentalis adults in bean leaf disk infested with two-spotted spider mites assessed as corrected mortality at 0.1167/46 g AI/L treated using worst-case laboratory exposure procedure measured after 72 hr
|
Metaseiulus occidentalis
|
42.4
%
|
|
Journal : Pest Manag Sci
Year : 2009
Volume : 65
Issue : 6
First Page : 635
Last Page : 639
|
Toxicity against Metaseiulus occidentalis adults in bean leaf disk infested with two-spotted spider mites assessed as corrected mortality at 0.1167/8 g AI/L treated using worst-case laboratory exposure procedure measured after 72 hr
|
Metaseiulus occidentalis
|
56.9
%
|
|
Journal : Pest Manag Sci
Year : 2009
Volume : 65
Issue : 6
First Page : 635
Last Page : 639
|
Toxicity against Metaseiulus occidentalis adults in bean leaf disk infested with two-spotted spider mites assessed as corrected mortality at 0.1167 g AI/L treated using worst-case laboratory exposure procedure measured after 72 hr
|
Metaseiulus occidentalis
|
63.7
%
|
|
Journal : Pest Manag Sci
Year : 2009
Volume : 65
Issue : 6
First Page : 635
Last Page : 639
|
Toxicity against Metaseiulus occidentalis adults in bean leaf disk infested with two-spotted spider mites assessed as corrected mortality at 2x0.1167 g AI/L treated using worst-case laboratory exposure procedure measured after 72 hr
|
Metaseiulus occidentalis
|
64.2
%
|
|
Journal : Pest Manag Sci
Year : 2009
Volume : 65
Issue : 6
First Page : 635
Last Page : 639
|
Toxicity against Metaseiulus occidentalis adults in bean leaf disk infested with two-spotted spider mites assessed as corrected mortality at 4x0.1167 g AI/L treated using worst-case laboratory exposure procedure measured after 72 hr
|
Metaseiulus occidentalis
|
83.1
%
|
|
Journal : Pest Manag Sci
Year : 2009
Volume : 65
Issue : 6
First Page : 635
Last Page : 639
|
Toxicity against Metaseiulus occidentalis adults in bean leaf disk infested with two-spotted spider mites assessed as mortality treated using worst-case laboratory exposure procedure measured after 72 hr
|
Metaseiulus occidentalis
|
0.021
gAi/L
|
|
Journal : Pest Manag Sci
Year : 2009
Volume : 65
Issue : 6
First Page : 635
Last Page : 639
|
Ovicidal activity against Metaseiulus occidentalis eggs in bean leaf disk infested with two-spotted spider mites assessed as corrected cumulative mortality at 700 g/kg WP formulation treated using worst-case laboratory exposure procedure measured after 144 hr (Rvb = 0.7%)
|
Metaseiulus occidentalis
|
2.0
%
|
|
Journal : Pest Manag Sci
Year : 2009
Volume : 65
Issue : 6
First Page : 635
Last Page : 639
|
Resistance ratio of LC50 for Bemisia tabaci biotype-B B4 (sweet potato whitefly) to LC50 for Bemisia tabaci LABS
|
Bemisia tabaci
|
15.0
|
|
Journal : Pest Manag Sci
Year : 2010
Volume : 66
Issue : 11
First Page : 1186
Last Page : 1190
|
Resistance ratio of LC50 for Bemisia tabaci biotype-B B3 (sweet potato whitefly) to LC50 for Bemisia tabaci LABS
|
Bemisia tabaci
|
30.0
|
|
Journal : Pest Manag Sci
Year : 2010
Volume : 66
Issue : 11
First Page : 1186
Last Page : 1190
|
Resistance ratio of LC50 for Bemisia tabaci biotype-B B2 (sweet potato whitefly) to LC50 for Bemisia tabaci LABS
|
Bemisia tabaci
|
1.2
|
|
Journal : Pest Manag Sci
Year : 2010
Volume : 66
Issue : 11
First Page : 1186
Last Page : 1190
|
Resistance ratio of LC50 for Bemisia tabaci biotype-B B1 (sweet potato whitefly) to LC50 for Bemisia tabaci LABS
|
Bemisia tabaci
|
2.1
|
|
Journal : Pest Manag Sci
Year : 2010
Volume : 66
Issue : 11
First Page : 1186
Last Page : 1190
|
Resistance ratio of LC50 for Bemisia tabaci biotype-Q Q3 (sweet potato whitefly) to LC50 for Bemisia tabaci LABS
|
Bemisia tabaci
|
57.0
|
|
Journal : Pest Manag Sci
Year : 2010
Volume : 66
Issue : 11
First Page : 1186
Last Page : 1190
|
Resistance ratio of LC50 for Bemisia tabaci biotype-Q Q2 (sweet potato whitefly) to LC50 for Bemisia tabaci LABS
|
Bemisia tabaci
|
20.0
|
|
Journal : Pest Manag Sci
Year : 2010
Volume : 66
Issue : 11
First Page : 1186
Last Page : 1190
|
Resistance ratio of LC50 for Bemisia tabaci biotype-Q Q1 (sweet potato whitefly) to LC50 for Bemisia tabaci LABS
|
Bemisia tabaci
|
0.95
|
|
Journal : Pest Manag Sci
Year : 2010
Volume : 66
Issue : 11
First Page : 1186
Last Page : 1190
|
Insecticidal activity against Bemisia tabaci biotype-B B4 (sweet potato whitefly) after 48 hr by leaf-dip bioassay
|
Bemisia tabaci
|
54.0
ug.mL-1
|
|
Journal : Pest Manag Sci
Year : 2010
Volume : 66
Issue : 11
First Page : 1186
Last Page : 1190
|
Insecticidal activity against Bemisia tabaci biotype-B B3 (sweet potato whitefly) after 48 hr by leaf-dip bioassay
|
Bemisia tabaci
|
110.0
ug.mL-1
|
|
Journal : Pest Manag Sci
Year : 2010
Volume : 66
Issue : 11
First Page : 1186
Last Page : 1190
|
Insecticidal activity against Bemisia tabaci biotype-B B2 (sweet potato whitefly) after 48 hr by leaf-dip bioassay
|
Bemisia tabaci
|
4.3
ug.mL-1
|
|
Journal : Pest Manag Sci
Year : 2010
Volume : 66
Issue : 11
First Page : 1186
Last Page : 1190
|
Insecticidal activity against Bemisia tabaci biotype-Q Q3 (sweet potato whitefly) after 48 hr by leaf-dip bioassay
|
Bemisia tabaci
|
210.0
ug.mL-1
|
|
Journal : Pest Manag Sci
Year : 2010
Volume : 66
Issue : 11
First Page : 1186
Last Page : 1190
|
Insecticidal activity against Bemisia tabaci biotype-B B1 (sweet potato whitefly) after 48 hr by leaf-dip bioassay
|
Bemisia tabaci
|
7.6
ug.mL-1
|
|
Journal : Pest Manag Sci
Year : 2010
Volume : 66
Issue : 11
First Page : 1186
Last Page : 1190
|
Insecticidal activity against Bemisia tabaci biotype-Q Q2 (sweet potato whitefly) after 48 hr by leaf-dip bioassay
|
Bemisia tabaci
|
74.0
ug.mL-1
|
|
Journal : Pest Manag Sci
Year : 2010
Volume : 66
Issue : 11
First Page : 1186
Last Page : 1190
|
Insecticidal activity against Bemisia tabaci LABS (sweet potato whitefly) after 48 hr by leaf-dip bioassay
|
Bemisia tabaci
|
3.7
ug.mL-1
|
|
Journal : Pest Manag Sci
Year : 2010
Volume : 66
Issue : 11
First Page : 1186
Last Page : 1190
|
Insecticidal activity against Bemisia tabaci biotype-Q Q1 (sweet potato whitefly) after 48 hr by leaf-dip bioassay
|
Bemisia tabaci
|
3.5
ug.mL-1
|
|
Journal : Pest Manag Sci
Year : 2010
Volume : 66
Issue : 11
First Page : 1186
Last Page : 1190
|
Insecticidal activity against fourth-instar larvae of Cydia pomonella (codling moth) lab-s compound applied topically measured after 4 days
|
Cydia pomonella
|
1.12
ug.mL-1
|
|
Journal : Pest Manag Sci
Year : 2012
Volume : 68
Issue : 6
First Page : 883
Last Page : 888
|
Insecticidal activity against Cydia pomonella (codling moth) lab-s neonate compound applied topically measured after 4 days
|
Cydia pomonella
|
0.26
ug.mL-1
|
|
Journal : Pest Manag Sci
Year : 2012
Volume : 68
Issue : 6
First Page : 883
Last Page : 888
|
Resistance index, ratio of LC50 for neonicotinoid-resistant Myzus persicae FRC-P (green peach aphid) to LC50 for Myzus persicae 4106A
|
Myzus persicae
|
82.0
|
|
Journal : Pest Manag Sci
Title : Investigating the mode of action of sulfoxaflor: a fourth-generation neonicotinoid.
Year : 2013
Volume : 69
Issue : 5
First Page : 607
Last Page : 619
Authors : Cutler P, Slater R, Edmunds AJ, Maienfisch P, Hall RG, Earley FG, Pitterna T, Pal S, Paul VL, Goodchild J, Blacker M, Hagmann L, Crossthwaite AJ.
Abstract : The precise mode of action of sulfoxaflor, a new nicotinic acetylcholine receptor-modulating insecticide, is unclear. A detailed understanding of the mode of action, especially in relation to the neonicotinoids, is essential for recommending effective pest management practices.Radiolabel binding experiments using a tritiated analogue of sulfoxaflor ([(3) H]-methyl-SFX) performed on membranes from Myzus persicae demonstrate that sulfoxaflor interacts specifically with the high-affinity imidacloprid binding site present in a subpopulation of the total nAChR pool. In competition studies, imidacloprid-like neonicotinoids displace [(3) H]-methyl-SFX at pM concentrations. The effects of sulfoxaflor on the exposed aphid nervous system in situ are analogous to those of imidacloprid and nitenpyram, and finally the high-affinity sulfoxaflor binding site is absent in a Myzus persicae strain (clone FRC) possessing a single amino acid point mutation (R81T) in the β-nAChR, a region critical for neonicotinoid interaction.The nicotinic acetylcholine receptor pharmacological profile of sulfoxaflor in aphids is consistent with that of imidacloprid. Additionally, the insecticidal activity of sulfoxaflor and the current commercialised neonicotinoids is affected by the point mutation in FRC Myzus persicae. Therefore, it is suggested that sulfoxalfor be considered a neonicotinoid, and that this be taken into account when recommending insecticide rotation partnering for effective resistance management programmes.
|
Insecticidal activity against Myzus persicae 4106A (green peach aphid) fed on compound treated chinese cabbage discs after 72 hr
|
Myzus persicae
|
0.6
ppm
|
|
Journal : Pest Manag Sci
Title : Investigating the mode of action of sulfoxaflor: a fourth-generation neonicotinoid.
Year : 2013
Volume : 69
Issue : 5
First Page : 607
Last Page : 619
Authors : Cutler P, Slater R, Edmunds AJ, Maienfisch P, Hall RG, Earley FG, Pitterna T, Pal S, Paul VL, Goodchild J, Blacker M, Hagmann L, Crossthwaite AJ.
Abstract : The precise mode of action of sulfoxaflor, a new nicotinic acetylcholine receptor-modulating insecticide, is unclear. A detailed understanding of the mode of action, especially in relation to the neonicotinoids, is essential for recommending effective pest management practices.Radiolabel binding experiments using a tritiated analogue of sulfoxaflor ([(3) H]-methyl-SFX) performed on membranes from Myzus persicae demonstrate that sulfoxaflor interacts specifically with the high-affinity imidacloprid binding site present in a subpopulation of the total nAChR pool. In competition studies, imidacloprid-like neonicotinoids displace [(3) H]-methyl-SFX at pM concentrations. The effects of sulfoxaflor on the exposed aphid nervous system in situ are analogous to those of imidacloprid and nitenpyram, and finally the high-affinity sulfoxaflor binding site is absent in a Myzus persicae strain (clone FRC) possessing a single amino acid point mutation (R81T) in the β-nAChR, a region critical for neonicotinoid interaction.The nicotinic acetylcholine receptor pharmacological profile of sulfoxaflor in aphids is consistent with that of imidacloprid. Additionally, the insecticidal activity of sulfoxaflor and the current commercialised neonicotinoids is affected by the point mutation in FRC Myzus persicae. Therefore, it is suggested that sulfoxalfor be considered a neonicotinoid, and that this be taken into account when recommending insecticide rotation partnering for effective resistance management programmes.
|
Insecticidal activity against neonicotinoid-resistant Myzus persicae FRC-P (green peach aphid) fed on compound treated chinese cabbage discs after 72 hr
|
Myzus persicae
|
49.3
ppm
|
|
Journal : Pest Manag Sci
Title : Investigating the mode of action of sulfoxaflor: a fourth-generation neonicotinoid.
Year : 2013
Volume : 69
Issue : 5
First Page : 607
Last Page : 619
Authors : Cutler P, Slater R, Edmunds AJ, Maienfisch P, Hall RG, Earley FG, Pitterna T, Pal S, Paul VL, Goodchild J, Blacker M, Hagmann L, Crossthwaite AJ.
Abstract : The precise mode of action of sulfoxaflor, a new nicotinic acetylcholine receptor-modulating insecticide, is unclear. A detailed understanding of the mode of action, especially in relation to the neonicotinoids, is essential for recommending effective pest management practices.Radiolabel binding experiments using a tritiated analogue of sulfoxaflor ([(3) H]-methyl-SFX) performed on membranes from Myzus persicae demonstrate that sulfoxaflor interacts specifically with the high-affinity imidacloprid binding site present in a subpopulation of the total nAChR pool. In competition studies, imidacloprid-like neonicotinoids displace [(3) H]-methyl-SFX at pM concentrations. The effects of sulfoxaflor on the exposed aphid nervous system in situ are analogous to those of imidacloprid and nitenpyram, and finally the high-affinity sulfoxaflor binding site is absent in a Myzus persicae strain (clone FRC) possessing a single amino acid point mutation (R81T) in the β-nAChR, a region critical for neonicotinoid interaction.The nicotinic acetylcholine receptor pharmacological profile of sulfoxaflor in aphids is consistent with that of imidacloprid. Additionally, the insecticidal activity of sulfoxaflor and the current commercialised neonicotinoids is affected by the point mutation in FRC Myzus persicae. Therefore, it is suggested that sulfoxalfor be considered a neonicotinoid, and that this be taken into account when recommending insecticide rotation partnering for effective resistance management programmes.
|
Lipophilicity, log P of the compound by HPLC based shake flask method
|
None
|
0.83
|
|
Journal : J Pesticide Sci
Year : 2003
Volume : 28
Issue : 1
First Page : 8
Last Page : 17
|
Neuroblocking activity in adult male Periplaneta americana (American cockroach) abdominal central nerve cords assessed as neuroblocking concentration measured at 22 to 25 degC
|
Periplaneta americana
|
6.19
|
|
Journal : J Pesticide Sci
Year : 2003
Volume : 28
Issue : 1
First Page : 8
Last Page : 17
|
Insecticidal activity against Musca domestica (house fly) dosed through injection into dorsal side of thorax assessed as mortality measured at intervals of 30 min for 2 hr
|
Musca domestica
|
426.58
nM
|
|
Journal : J Pesticide Sci
Year : 2003
Volume : 28
Issue : 1
First Page : 8
Last Page : 17
|
Insecticidal activity against adult male Periplaneta americana (American cockroach) dosed through injection into abdomen assessed as mortality measured 24 hr post dose in presence of 50 ug metabolic inhibitor PB and 50 ug NIA
|
Periplaneta americana
|
9.68
|
|
Journal : J Pesticide Sci
Year : 2003
Volume : 28
Issue : 1
First Page : 8
Last Page : 17
|
Insecticidal activity against adult male Periplaneta americana (American cockroach) dosed through injection into abdomen assessed as mortality measured 24 hr post dose in presence of 50 ug metabolic inhibitor PB
|
Periplaneta americana
|
8.58
|
|
Journal : J Pesticide Sci
Year : 2003
Volume : 28
Issue : 1
First Page : 8
Last Page : 17
|
Insecticidal activity against adult male Periplaneta americana (American cockroach) dosed through injection into abdomen assessed as mortality measured 24 hr post dose
|
Periplaneta americana
|
8.28
|
|
Journal : J Pesticide Sci
Year : 2003
Volume : 28
Issue : 1
First Page : 8
Last Page : 17
|
Insecticidal activity against imidacloprid-resistant Aphis gossypii (cotton aphid) in cotton leaves assessed as fecundity at LC20 concentration
|
Aphis gossypii
|
None
|
|
Journal : Pest Manag Sci
Year : 2011
Volume : 67
Issue : 12
First Page : 1528
Last Page : 1533
|
Insecticidal activity against imidacloprid-resistant Aphis gossypii (cotton aphid) in cotton leaves assessed as longevity at LC20 concentration
|
Aphis gossypii
|
11.4
day
|
|
Journal : Pest Manag Sci
Year : 2011
Volume : 67
Issue : 12
First Page : 1528
Last Page : 1533
|
Insecticidal activity against imidacloprid-resistant Aphis gossypii (cotton aphid) in cotton leaves assessed as inhibition of honeydew excretion at LC20 concentration measured after 72 hr by dipping method
|
Aphis gossypii
|
40.2
%
|
|
Journal : Pest Manag Sci
Year : 2011
Volume : 67
Issue : 12
First Page : 1528
Last Page : 1533
|
Insecticidal activity against imidacloprid-resistant Aphis gossypii (cotton aphid) in cotton leaves assessed as reduction in body weight at LC20 concentration measured after 72 hr by dipping method (Rvb = 0.40 +/- 0.0002 mg/aphid)
|
Aphis gossypii
|
0.16
mg
|
|
Journal : Pest Manag Sci
Year : 2011
Volume : 67
Issue : 12
First Page : 1528
Last Page : 1533
|
Insecticidal activity against imidacloprid-resistant Aphis gossypii (cotton aphid) in cotton leaves assessed as reduction in body weight at LC20 concentration measured after 48 hr by dipping method (Rvb = 0.36 +/- 0.0006 mg/aphid)
|
Aphis gossypii
|
0.18
mg
|
|
Journal : Pest Manag Sci
Year : 2011
Volume : 67
Issue : 12
First Page : 1528
Last Page : 1533
|
Insecticidal activity against imidacloprid-resistant Aphis gossypii (cotton aphid) in cotton leaves assessed as mortality measured after 72 hr by dipping method
|
Aphis gossypii
|
8.36
ug ml-1
|
|
Journal : Pest Manag Sci
Year : 2011
Volume : 67
Issue : 12
First Page : 1528
Last Page : 1533
|
Insecticidal activity against imidacloprid-resistant Aphis gossypii (cotton aphid) in cotton leaves assessed as reduction in body weight at LC20 concentration measured after 24 hr by dipping method (Rvb = 0.33 +/- 0.0004 mg/aphid)
|
Aphis gossypii
|
0.21
mg
|
|
Journal : Pest Manag Sci
Year : 2011
Volume : 67
Issue : 12
First Page : 1528
Last Page : 1533
|
Resistance ratio, LC50 for Aphis gossypii RF45 (cotton aphid) to LC50 for Aphis gossypii SS
|
Aphis gossypii
|
4.52
|
|
Journal : Pest Manag Sci
Year : 2011
Volume : 67
Issue : 12
First Page : 1528
Last Page : 1533
|
Insecticidal activity against imidacloprid-resistant Aphis gossypii RF45 (cotton aphid) in cotton leaves assessed as mortality measured after 48 hr by dipping method
|
Aphis gossypii
|
12.4
microg/mL2
|
|
Journal : Pest Manag Sci
Year : 2011
Volume : 67
Issue : 12
First Page : 1528
Last Page : 1533
|
Insecticidal activity against Aphis gossypii SS (cotton aphid) in cotton leaves assessed as mortality measured after 48 hr by dipping method
|
Aphis gossypii
|
2.73
microg/mL2
|
|
Journal : Pest Manag Sci
Year : 2011
Volume : 67
Issue : 12
First Page : 1528
Last Page : 1533
|
Displacement of [3H]alpha-BGT from nAChR in Torpedo nobiliana electric organs at 300 uM
|
Torpedo nobiliana
|
12.6
%
|
|
Journal : J Pesticide Sci
Title : Molecular Mechanism for Selective Toxicity of Nicotinoids and Neonicotinoids
Year : 1995
Volume : 20
Issue : 1
First Page : 33
Last Page : 40
Authors : YAMAMOTO I, YABUTA G, TOMIZAWA M, SAITO T, MIYAMOTO T, KAGABU S
|
Displacement of [3H]alpha-BGT from nAChR in Apis mellifera (honeybee) head
|
Apis mellifera
|
8830.0
nM
|
|
Journal : J Pesticide Sci
Title : Molecular Mechanism for Selective Toxicity of Nicotinoids and Neonicotinoids
Year : 1995
Volume : 20
Issue : 1
First Page : 33
Last Page : 40
Authors : YAMAMOTO I, YABUTA G, TOMIZAWA M, SAITO T, MIYAMOTO T, KAGABU S
|
Displacement of [3H]alpha-BGT from nAChR in Torpedo nobiliana electric organs
|
Torpedo nobiliana
|
300000.0
nM
|
|
Journal : J Pesticide Sci
Title : Molecular Mechanism for Selective Toxicity of Nicotinoids and Neonicotinoids
Year : 1995
Volume : 20
Issue : 1
First Page : 33
Last Page : 40
Authors : YAMAMOTO I, YABUTA G, TOMIZAWA M, SAITO T, MIYAMOTO T, KAGABU S
|
Resistant factor, ratio of LC50 for Aphis gossypii clone Ivo (cotton aphid) to Aphis gossypii clone NM1
|
Aphis gossypii
|
1.0
|
|
Journal : Pest Manag Sci
Title : Insecticide resistance traits differ among and within host races in Aphis gossypii.
Year : 2010
Volume : 66
Issue : 3
First Page : 301
Last Page : 307
Authors : Carletto J, Martin T, Vanlerberghe-Masutti F, Brévault T.
Abstract : BACKGROUND: The polyphagous cotton-melon aphid Aphis gossypii Glover is structured into geographically widespread host races comprising a few clones specialised on Cucurbitaceae, cotton, eggplant or pepper. To assess insecticide resistance among and within host races, leaf disc bioassays were conducted on aphid clones collected from Cucurbitaceae (genotypes C4 and C9), cotton (genotypes Burk and Ivo), eggplant (genotype Auber) and pepper (genotype PsP4). Molecular diagnostic (PCR-RFLP) and enzyme assays were also performed to detect the basic mechanisms underlying insecticide resistance. RESULTS: All six clones were susceptible to acetamiprid (neonicotinoid) or carbosulfan (carbamate). Conversely, all clones were resistant to dimethoate (organophosphate) (RF = 4.1-38.1) and carried mutation S431F in the acetylcholinesterase gene. Auber, PsP4 and Burk also carried mutation A302S in this gene, which possibly conferred moderate resistance (RF = 3.7-6.8) to profenofos and monocrotophos (organophosphates). Auber and Burk were highly resistant (RF = 41.2 and 473 respectively) to cypermethrin (pyrethroid). This resistance was likely associated with point mutation super-kdr (M918L) in the voltage-gated sodium channel gene (para gene) or metabolic detoxification mediated by esterase and oxidase enzymes. CONCLUSION: Multiple resistance to a broad range of insecticides and multiple mechanisms of resistance in some clones could explain to some extent the low genetic diversity observed within A. gossypii host races.
|
Resistant factor, ratio of LC50 for Aphis gossypii clone Burk (cotton aphid) to Aphis gossypii clone NM1
|
Aphis gossypii
|
1.6
|
|
Journal : Pest Manag Sci
Title : Insecticide resistance traits differ among and within host races in Aphis gossypii.
Year : 2010
Volume : 66
Issue : 3
First Page : 301
Last Page : 307
Authors : Carletto J, Martin T, Vanlerberghe-Masutti F, Brévault T.
Abstract : BACKGROUND: The polyphagous cotton-melon aphid Aphis gossypii Glover is structured into geographically widespread host races comprising a few clones specialised on Cucurbitaceae, cotton, eggplant or pepper. To assess insecticide resistance among and within host races, leaf disc bioassays were conducted on aphid clones collected from Cucurbitaceae (genotypes C4 and C9), cotton (genotypes Burk and Ivo), eggplant (genotype Auber) and pepper (genotype PsP4). Molecular diagnostic (PCR-RFLP) and enzyme assays were also performed to detect the basic mechanisms underlying insecticide resistance. RESULTS: All six clones were susceptible to acetamiprid (neonicotinoid) or carbosulfan (carbamate). Conversely, all clones were resistant to dimethoate (organophosphate) (RF = 4.1-38.1) and carried mutation S431F in the acetylcholinesterase gene. Auber, PsP4 and Burk also carried mutation A302S in this gene, which possibly conferred moderate resistance (RF = 3.7-6.8) to profenofos and monocrotophos (organophosphates). Auber and Burk were highly resistant (RF = 41.2 and 473 respectively) to cypermethrin (pyrethroid). This resistance was likely associated with point mutation super-kdr (M918L) in the voltage-gated sodium channel gene (para gene) or metabolic detoxification mediated by esterase and oxidase enzymes. CONCLUSION: Multiple resistance to a broad range of insecticides and multiple mechanisms of resistance in some clones could explain to some extent the low genetic diversity observed within A. gossypii host races.
|
Resistant factor, ratio of LC50 for Aphis gossypii clone Auber (cotton aphid) to Aphis gossypii clone NM1
|
Aphis gossypii
|
2.0
|
|
Journal : Pest Manag Sci
Title : Insecticide resistance traits differ among and within host races in Aphis gossypii.
Year : 2010
Volume : 66
Issue : 3
First Page : 301
Last Page : 307
Authors : Carletto J, Martin T, Vanlerberghe-Masutti F, Brévault T.
Abstract : BACKGROUND: The polyphagous cotton-melon aphid Aphis gossypii Glover is structured into geographically widespread host races comprising a few clones specialised on Cucurbitaceae, cotton, eggplant or pepper. To assess insecticide resistance among and within host races, leaf disc bioassays were conducted on aphid clones collected from Cucurbitaceae (genotypes C4 and C9), cotton (genotypes Burk and Ivo), eggplant (genotype Auber) and pepper (genotype PsP4). Molecular diagnostic (PCR-RFLP) and enzyme assays were also performed to detect the basic mechanisms underlying insecticide resistance. RESULTS: All six clones were susceptible to acetamiprid (neonicotinoid) or carbosulfan (carbamate). Conversely, all clones were resistant to dimethoate (organophosphate) (RF = 4.1-38.1) and carried mutation S431F in the acetylcholinesterase gene. Auber, PsP4 and Burk also carried mutation A302S in this gene, which possibly conferred moderate resistance (RF = 3.7-6.8) to profenofos and monocrotophos (organophosphates). Auber and Burk were highly resistant (RF = 41.2 and 473 respectively) to cypermethrin (pyrethroid). This resistance was likely associated with point mutation super-kdr (M918L) in the voltage-gated sodium channel gene (para gene) or metabolic detoxification mediated by esterase and oxidase enzymes. CONCLUSION: Multiple resistance to a broad range of insecticides and multiple mechanisms of resistance in some clones could explain to some extent the low genetic diversity observed within A. gossypii host races.
|
Resistant factor, ratio of LC50 for Aphis gossypii clone PsP4 (cotton aphid) to Aphis gossypii clone NM1
|
Aphis gossypii
|
0.6
|
|
Journal : Pest Manag Sci
Title : Insecticide resistance traits differ among and within host races in Aphis gossypii.
Year : 2010
Volume : 66
Issue : 3
First Page : 301
Last Page : 307
Authors : Carletto J, Martin T, Vanlerberghe-Masutti F, Brévault T.
Abstract : BACKGROUND: The polyphagous cotton-melon aphid Aphis gossypii Glover is structured into geographically widespread host races comprising a few clones specialised on Cucurbitaceae, cotton, eggplant or pepper. To assess insecticide resistance among and within host races, leaf disc bioassays were conducted on aphid clones collected from Cucurbitaceae (genotypes C4 and C9), cotton (genotypes Burk and Ivo), eggplant (genotype Auber) and pepper (genotype PsP4). Molecular diagnostic (PCR-RFLP) and enzyme assays were also performed to detect the basic mechanisms underlying insecticide resistance. RESULTS: All six clones were susceptible to acetamiprid (neonicotinoid) or carbosulfan (carbamate). Conversely, all clones were resistant to dimethoate (organophosphate) (RF = 4.1-38.1) and carried mutation S431F in the acetylcholinesterase gene. Auber, PsP4 and Burk also carried mutation A302S in this gene, which possibly conferred moderate resistance (RF = 3.7-6.8) to profenofos and monocrotophos (organophosphates). Auber and Burk were highly resistant (RF = 41.2 and 473 respectively) to cypermethrin (pyrethroid). This resistance was likely associated with point mutation super-kdr (M918L) in the voltage-gated sodium channel gene (para gene) or metabolic detoxification mediated by esterase and oxidase enzymes. CONCLUSION: Multiple resistance to a broad range of insecticides and multiple mechanisms of resistance in some clones could explain to some extent the low genetic diversity observed within A. gossypii host races.
|
Resistant factor, ratio of LC50 for Aphis gossypii clone C4 (cotton aphid) to Aphis gossypii clone NM1
|
Aphis gossypii
|
0.9
|
|
Journal : Pest Manag Sci
Title : Insecticide resistance traits differ among and within host races in Aphis gossypii.
Year : 2010
Volume : 66
Issue : 3
First Page : 301
Last Page : 307
Authors : Carletto J, Martin T, Vanlerberghe-Masutti F, Brévault T.
Abstract : BACKGROUND: The polyphagous cotton-melon aphid Aphis gossypii Glover is structured into geographically widespread host races comprising a few clones specialised on Cucurbitaceae, cotton, eggplant or pepper. To assess insecticide resistance among and within host races, leaf disc bioassays were conducted on aphid clones collected from Cucurbitaceae (genotypes C4 and C9), cotton (genotypes Burk and Ivo), eggplant (genotype Auber) and pepper (genotype PsP4). Molecular diagnostic (PCR-RFLP) and enzyme assays were also performed to detect the basic mechanisms underlying insecticide resistance. RESULTS: All six clones were susceptible to acetamiprid (neonicotinoid) or carbosulfan (carbamate). Conversely, all clones were resistant to dimethoate (organophosphate) (RF = 4.1-38.1) and carried mutation S431F in the acetylcholinesterase gene. Auber, PsP4 and Burk also carried mutation A302S in this gene, which possibly conferred moderate resistance (RF = 3.7-6.8) to profenofos and monocrotophos (organophosphates). Auber and Burk were highly resistant (RF = 41.2 and 473 respectively) to cypermethrin (pyrethroid). This resistance was likely associated with point mutation super-kdr (M918L) in the voltage-gated sodium channel gene (para gene) or metabolic detoxification mediated by esterase and oxidase enzymes. CONCLUSION: Multiple resistance to a broad range of insecticides and multiple mechanisms of resistance in some clones could explain to some extent the low genetic diversity observed within A. gossypii host races.
|
Resistant factor, ratio of LC50 for Aphis gossypii clone C9 (cotton aphid) to Aphis gossypii clone NM1
|
Aphis gossypii
|
1.1
|
|
Journal : Pest Manag Sci
Title : Insecticide resistance traits differ among and within host races in Aphis gossypii.
Year : 2010
Volume : 66
Issue : 3
First Page : 301
Last Page : 307
Authors : Carletto J, Martin T, Vanlerberghe-Masutti F, Brévault T.
Abstract : BACKGROUND: The polyphagous cotton-melon aphid Aphis gossypii Glover is structured into geographically widespread host races comprising a few clones specialised on Cucurbitaceae, cotton, eggplant or pepper. To assess insecticide resistance among and within host races, leaf disc bioassays were conducted on aphid clones collected from Cucurbitaceae (genotypes C4 and C9), cotton (genotypes Burk and Ivo), eggplant (genotype Auber) and pepper (genotype PsP4). Molecular diagnostic (PCR-RFLP) and enzyme assays were also performed to detect the basic mechanisms underlying insecticide resistance. RESULTS: All six clones were susceptible to acetamiprid (neonicotinoid) or carbosulfan (carbamate). Conversely, all clones were resistant to dimethoate (organophosphate) (RF = 4.1-38.1) and carried mutation S431F in the acetylcholinesterase gene. Auber, PsP4 and Burk also carried mutation A302S in this gene, which possibly conferred moderate resistance (RF = 3.7-6.8) to profenofos and monocrotophos (organophosphates). Auber and Burk were highly resistant (RF = 41.2 and 473 respectively) to cypermethrin (pyrethroid). This resistance was likely associated with point mutation super-kdr (M918L) in the voltage-gated sodium channel gene (para gene) or metabolic detoxification mediated by esterase and oxidase enzymes. CONCLUSION: Multiple resistance to a broad range of insecticides and multiple mechanisms of resistance in some clones could explain to some extent the low genetic diversity observed within A. gossypii host races.
|
Insecticidal activity against Aphis gossypii clone Ivo (cotton aphid) in cotton plant leaf assessed as mortality after 24 hr by leaf disk assay
|
Aphis gossypii
|
4.0
ugAi/ml
|
|
Journal : Pest Manag Sci
Title : Insecticide resistance traits differ among and within host races in Aphis gossypii.
Year : 2010
Volume : 66
Issue : 3
First Page : 301
Last Page : 307
Authors : Carletto J, Martin T, Vanlerberghe-Masutti F, Brévault T.
Abstract : BACKGROUND: The polyphagous cotton-melon aphid Aphis gossypii Glover is structured into geographically widespread host races comprising a few clones specialised on Cucurbitaceae, cotton, eggplant or pepper. To assess insecticide resistance among and within host races, leaf disc bioassays were conducted on aphid clones collected from Cucurbitaceae (genotypes C4 and C9), cotton (genotypes Burk and Ivo), eggplant (genotype Auber) and pepper (genotype PsP4). Molecular diagnostic (PCR-RFLP) and enzyme assays were also performed to detect the basic mechanisms underlying insecticide resistance. RESULTS: All six clones were susceptible to acetamiprid (neonicotinoid) or carbosulfan (carbamate). Conversely, all clones were resistant to dimethoate (organophosphate) (RF = 4.1-38.1) and carried mutation S431F in the acetylcholinesterase gene. Auber, PsP4 and Burk also carried mutation A302S in this gene, which possibly conferred moderate resistance (RF = 3.7-6.8) to profenofos and monocrotophos (organophosphates). Auber and Burk were highly resistant (RF = 41.2 and 473 respectively) to cypermethrin (pyrethroid). This resistance was likely associated with point mutation super-kdr (M918L) in the voltage-gated sodium channel gene (para gene) or metabolic detoxification mediated by esterase and oxidase enzymes. CONCLUSION: Multiple resistance to a broad range of insecticides and multiple mechanisms of resistance in some clones could explain to some extent the low genetic diversity observed within A. gossypii host races.
|
Insecticidal activity against Aphis gossypii clone Burk (cotton aphid) in cotton plant leaf assessed as mortality after 24 hr by leaf disk assay
|
Aphis gossypii
|
6.4
ugAi/ml
|
|
Journal : Pest Manag Sci
Title : Insecticide resistance traits differ among and within host races in Aphis gossypii.
Year : 2010
Volume : 66
Issue : 3
First Page : 301
Last Page : 307
Authors : Carletto J, Martin T, Vanlerberghe-Masutti F, Brévault T.
Abstract : BACKGROUND: The polyphagous cotton-melon aphid Aphis gossypii Glover is structured into geographically widespread host races comprising a few clones specialised on Cucurbitaceae, cotton, eggplant or pepper. To assess insecticide resistance among and within host races, leaf disc bioassays were conducted on aphid clones collected from Cucurbitaceae (genotypes C4 and C9), cotton (genotypes Burk and Ivo), eggplant (genotype Auber) and pepper (genotype PsP4). Molecular diagnostic (PCR-RFLP) and enzyme assays were also performed to detect the basic mechanisms underlying insecticide resistance. RESULTS: All six clones were susceptible to acetamiprid (neonicotinoid) or carbosulfan (carbamate). Conversely, all clones were resistant to dimethoate (organophosphate) (RF = 4.1-38.1) and carried mutation S431F in the acetylcholinesterase gene. Auber, PsP4 and Burk also carried mutation A302S in this gene, which possibly conferred moderate resistance (RF = 3.7-6.8) to profenofos and monocrotophos (organophosphates). Auber and Burk were highly resistant (RF = 41.2 and 473 respectively) to cypermethrin (pyrethroid). This resistance was likely associated with point mutation super-kdr (M918L) in the voltage-gated sodium channel gene (para gene) or metabolic detoxification mediated by esterase and oxidase enzymes. CONCLUSION: Multiple resistance to a broad range of insecticides and multiple mechanisms of resistance in some clones could explain to some extent the low genetic diversity observed within A. gossypii host races.
|
Insecticidal activity against Aphis gossypii clone Auber (cotton aphid) in egg plant leaf assessed as mortality after 24 hr by leaf disk assay
|
Aphis gossypii
|
7.9
ugAi/ml
|
|
Journal : Pest Manag Sci
Title : Insecticide resistance traits differ among and within host races in Aphis gossypii.
Year : 2010
Volume : 66
Issue : 3
First Page : 301
Last Page : 307
Authors : Carletto J, Martin T, Vanlerberghe-Masutti F, Brévault T.
Abstract : BACKGROUND: The polyphagous cotton-melon aphid Aphis gossypii Glover is structured into geographically widespread host races comprising a few clones specialised on Cucurbitaceae, cotton, eggplant or pepper. To assess insecticide resistance among and within host races, leaf disc bioassays were conducted on aphid clones collected from Cucurbitaceae (genotypes C4 and C9), cotton (genotypes Burk and Ivo), eggplant (genotype Auber) and pepper (genotype PsP4). Molecular diagnostic (PCR-RFLP) and enzyme assays were also performed to detect the basic mechanisms underlying insecticide resistance. RESULTS: All six clones were susceptible to acetamiprid (neonicotinoid) or carbosulfan (carbamate). Conversely, all clones were resistant to dimethoate (organophosphate) (RF = 4.1-38.1) and carried mutation S431F in the acetylcholinesterase gene. Auber, PsP4 and Burk also carried mutation A302S in this gene, which possibly conferred moderate resistance (RF = 3.7-6.8) to profenofos and monocrotophos (organophosphates). Auber and Burk were highly resistant (RF = 41.2 and 473 respectively) to cypermethrin (pyrethroid). This resistance was likely associated with point mutation super-kdr (M918L) in the voltage-gated sodium channel gene (para gene) or metabolic detoxification mediated by esterase and oxidase enzymes. CONCLUSION: Multiple resistance to a broad range of insecticides and multiple mechanisms of resistance in some clones could explain to some extent the low genetic diversity observed within A. gossypii host races.
|
Insecticidal activity against Aphis gossypii clone PsP4 (cotton aphid) in pepper plant leaf assessed as mortality after 24 hr by leaf disk assay
|
Aphis gossypii
|
2.5
ugAi/ml
|
|
Journal : Pest Manag Sci
Title : Insecticide resistance traits differ among and within host races in Aphis gossypii.
Year : 2010
Volume : 66
Issue : 3
First Page : 301
Last Page : 307
Authors : Carletto J, Martin T, Vanlerberghe-Masutti F, Brévault T.
Abstract : BACKGROUND: The polyphagous cotton-melon aphid Aphis gossypii Glover is structured into geographically widespread host races comprising a few clones specialised on Cucurbitaceae, cotton, eggplant or pepper. To assess insecticide resistance among and within host races, leaf disc bioassays were conducted on aphid clones collected from Cucurbitaceae (genotypes C4 and C9), cotton (genotypes Burk and Ivo), eggplant (genotype Auber) and pepper (genotype PsP4). Molecular diagnostic (PCR-RFLP) and enzyme assays were also performed to detect the basic mechanisms underlying insecticide resistance. RESULTS: All six clones were susceptible to acetamiprid (neonicotinoid) or carbosulfan (carbamate). Conversely, all clones were resistant to dimethoate (organophosphate) (RF = 4.1-38.1) and carried mutation S431F in the acetylcholinesterase gene. Auber, PsP4 and Burk also carried mutation A302S in this gene, which possibly conferred moderate resistance (RF = 3.7-6.8) to profenofos and monocrotophos (organophosphates). Auber and Burk were highly resistant (RF = 41.2 and 473 respectively) to cypermethrin (pyrethroid). This resistance was likely associated with point mutation super-kdr (M918L) in the voltage-gated sodium channel gene (para gene) or metabolic detoxification mediated by esterase and oxidase enzymes. CONCLUSION: Multiple resistance to a broad range of insecticides and multiple mechanisms of resistance in some clones could explain to some extent the low genetic diversity observed within A. gossypii host races.
|
Insecticidal activity against Aphis gossypii clone C4 (cotton aphid) in zucchini or squash leaf assessed as mortality after 24 hr by leaf disk assay
|
Aphis gossypii
|
3.7
ugAi/ml
|
|
Journal : Pest Manag Sci
Title : Insecticide resistance traits differ among and within host races in Aphis gossypii.
Year : 2010
Volume : 66
Issue : 3
First Page : 301
Last Page : 307
Authors : Carletto J, Martin T, Vanlerberghe-Masutti F, Brévault T.
Abstract : BACKGROUND: The polyphagous cotton-melon aphid Aphis gossypii Glover is structured into geographically widespread host races comprising a few clones specialised on Cucurbitaceae, cotton, eggplant or pepper. To assess insecticide resistance among and within host races, leaf disc bioassays were conducted on aphid clones collected from Cucurbitaceae (genotypes C4 and C9), cotton (genotypes Burk and Ivo), eggplant (genotype Auber) and pepper (genotype PsP4). Molecular diagnostic (PCR-RFLP) and enzyme assays were also performed to detect the basic mechanisms underlying insecticide resistance. RESULTS: All six clones were susceptible to acetamiprid (neonicotinoid) or carbosulfan (carbamate). Conversely, all clones were resistant to dimethoate (organophosphate) (RF = 4.1-38.1) and carried mutation S431F in the acetylcholinesterase gene. Auber, PsP4 and Burk also carried mutation A302S in this gene, which possibly conferred moderate resistance (RF = 3.7-6.8) to profenofos and monocrotophos (organophosphates). Auber and Burk were highly resistant (RF = 41.2 and 473 respectively) to cypermethrin (pyrethroid). This resistance was likely associated with point mutation super-kdr (M918L) in the voltage-gated sodium channel gene (para gene) or metabolic detoxification mediated by esterase and oxidase enzymes. CONCLUSION: Multiple resistance to a broad range of insecticides and multiple mechanisms of resistance in some clones could explain to some extent the low genetic diversity observed within A. gossypii host races.
|
Insecticidal activity against Aphis gossypii clone C9 (cotton aphid) in zucchini or squash leaf assessed as mortality after 24 hr by leaf disk assay
|
Aphis gossypii
|
4.3
ugAi/ml
|
|
Journal : Pest Manag Sci
Title : Insecticide resistance traits differ among and within host races in Aphis gossypii.
Year : 2010
Volume : 66
Issue : 3
First Page : 301
Last Page : 307
Authors : Carletto J, Martin T, Vanlerberghe-Masutti F, Brévault T.
Abstract : BACKGROUND: The polyphagous cotton-melon aphid Aphis gossypii Glover is structured into geographically widespread host races comprising a few clones specialised on Cucurbitaceae, cotton, eggplant or pepper. To assess insecticide resistance among and within host races, leaf disc bioassays were conducted on aphid clones collected from Cucurbitaceae (genotypes C4 and C9), cotton (genotypes Burk and Ivo), eggplant (genotype Auber) and pepper (genotype PsP4). Molecular diagnostic (PCR-RFLP) and enzyme assays were also performed to detect the basic mechanisms underlying insecticide resistance. RESULTS: All six clones were susceptible to acetamiprid (neonicotinoid) or carbosulfan (carbamate). Conversely, all clones were resistant to dimethoate (organophosphate) (RF = 4.1-38.1) and carried mutation S431F in the acetylcholinesterase gene. Auber, PsP4 and Burk also carried mutation A302S in this gene, which possibly conferred moderate resistance (RF = 3.7-6.8) to profenofos and monocrotophos (organophosphates). Auber and Burk were highly resistant (RF = 41.2 and 473 respectively) to cypermethrin (pyrethroid). This resistance was likely associated with point mutation super-kdr (M918L) in the voltage-gated sodium channel gene (para gene) or metabolic detoxification mediated by esterase and oxidase enzymes. CONCLUSION: Multiple resistance to a broad range of insecticides and multiple mechanisms of resistance in some clones could explain to some extent the low genetic diversity observed within A. gossypii host races.
|
Insecticidal activity against Aphis gossypii clone NM1 (cotton aphid) in zucchini or squash leaf assessed as mortality after 24 hr by leaf disk assay
|
Aphis gossypii
|
3.9
ugAi/ml
|
|
Journal : Pest Manag Sci
Title : Insecticide resistance traits differ among and within host races in Aphis gossypii.
Year : 2010
Volume : 66
Issue : 3
First Page : 301
Last Page : 307
Authors : Carletto J, Martin T, Vanlerberghe-Masutti F, Brévault T.
Abstract : BACKGROUND: The polyphagous cotton-melon aphid Aphis gossypii Glover is structured into geographically widespread host races comprising a few clones specialised on Cucurbitaceae, cotton, eggplant or pepper. To assess insecticide resistance among and within host races, leaf disc bioassays were conducted on aphid clones collected from Cucurbitaceae (genotypes C4 and C9), cotton (genotypes Burk and Ivo), eggplant (genotype Auber) and pepper (genotype PsP4). Molecular diagnostic (PCR-RFLP) and enzyme assays were also performed to detect the basic mechanisms underlying insecticide resistance. RESULTS: All six clones were susceptible to acetamiprid (neonicotinoid) or carbosulfan (carbamate). Conversely, all clones were resistant to dimethoate (organophosphate) (RF = 4.1-38.1) and carried mutation S431F in the acetylcholinesterase gene. Auber, PsP4 and Burk also carried mutation A302S in this gene, which possibly conferred moderate resistance (RF = 3.7-6.8) to profenofos and monocrotophos (organophosphates). Auber and Burk were highly resistant (RF = 41.2 and 473 respectively) to cypermethrin (pyrethroid). This resistance was likely associated with point mutation super-kdr (M918L) in the voltage-gated sodium channel gene (para gene) or metabolic detoxification mediated by esterase and oxidase enzymes. CONCLUSION: Multiple resistance to a broad range of insecticides and multiple mechanisms of resistance in some clones could explain to some extent the low genetic diversity observed within A. gossypii host races.
|
Cross resistant, ratio of LC50 for B-biotype thiamethoxam-resistant Bemisia tabaci (sweet potato whitefly) to LC50 for B-biotype thiamethoxam-sensitive Bemisia tabaci (sweet potato whitefly)
|
Bemisia tabaci
|
35.8
|
|
Journal : Pest Manag Sci
Title : Cross-resistance study and biochemical mechanisms of thiamethoxam resistance in B-biotype Bemisia tabaci (Hemiptera: Aleyrodidae).
Year : 2010
Volume : 66
Issue : 3
First Page : 313
Last Page : 318
Authors : Feng Y, Wu Q, Wang S, Chang X, Xie W, Xu B, Zhang Y.
Abstract : BACKGROUND: B-biotype Bemisia tabaci (Gennadius) has invaded China over the past two decades. To understand the risks and to determine possible mechanisms of resistance to thiamethoxam in B. tabaci, a resistant strain was selected in the laboratory. Cross-resistance and the biochemical mechanisms of thiamethoxam resistance were investigated in the present study. RESULTS: A 66.3-fold thiamethoxam-resistant B. tabaci strain (TH-R) was established after selection for 36 generations. Compared with the susceptible strain (TH-S), the selected TH-R strain showed obvious cross-resistance to imidacloprid (47.3-fold), acetamiprid (35.8-fold), nitenpyram (9.99-fold), abamectin (5.33-fold) and carbosulfan (4.43-fold). No cross-resistance to fipronil, chlorpyrifos or deltamethrin was seen. Piperonyl butoxide (PBO) and triphenyl phosphate (TPP) exhibited significant synergism on thiamethoxam effects in the TH-R strain (3.14- and 2.37-fold respectively). However, diethyl maleate (DEM) did not act synergistically with thiamethoxam. Biochemical assays showed that cytochrome P450 monooxygenase activities increased 1.21- and 1.68-fold respectively, and carboxylesterase activity increased 2.96-fold in the TH-R strain. However, no difference was observed for glutathione S-transferase between the two strains. CONCLUSION: B-biotype B. tabaci develops resistance to thiamethoxam. Cytochrome P450 monooxygenase and carboxylesterase appear to be responsible for the resistance. Reasonable resistance management that avoids the use of cross-resistance insecticides may delay the development of resistance to thiamethoxam in this species.
|
Insecticidal activity against B-biotype thiamethoxam-resistant Bemisia tabaci (sweet potato whitefly) in cabbage leaf assessed as mortality after 48 hr by leaf disk assay
|
Bemisia tabaci
|
70.9
ug.mL-1
|
|
Journal : Pest Manag Sci
Title : Cross-resistance study and biochemical mechanisms of thiamethoxam resistance in B-biotype Bemisia tabaci (Hemiptera: Aleyrodidae).
Year : 2010
Volume : 66
Issue : 3
First Page : 313
Last Page : 318
Authors : Feng Y, Wu Q, Wang S, Chang X, Xie W, Xu B, Zhang Y.
Abstract : BACKGROUND: B-biotype Bemisia tabaci (Gennadius) has invaded China over the past two decades. To understand the risks and to determine possible mechanisms of resistance to thiamethoxam in B. tabaci, a resistant strain was selected in the laboratory. Cross-resistance and the biochemical mechanisms of thiamethoxam resistance were investigated in the present study. RESULTS: A 66.3-fold thiamethoxam-resistant B. tabaci strain (TH-R) was established after selection for 36 generations. Compared with the susceptible strain (TH-S), the selected TH-R strain showed obvious cross-resistance to imidacloprid (47.3-fold), acetamiprid (35.8-fold), nitenpyram (9.99-fold), abamectin (5.33-fold) and carbosulfan (4.43-fold). No cross-resistance to fipronil, chlorpyrifos or deltamethrin was seen. Piperonyl butoxide (PBO) and triphenyl phosphate (TPP) exhibited significant synergism on thiamethoxam effects in the TH-R strain (3.14- and 2.37-fold respectively). However, diethyl maleate (DEM) did not act synergistically with thiamethoxam. Biochemical assays showed that cytochrome P450 monooxygenase activities increased 1.21- and 1.68-fold respectively, and carboxylesterase activity increased 2.96-fold in the TH-R strain. However, no difference was observed for glutathione S-transferase between the two strains. CONCLUSION: B-biotype B. tabaci develops resistance to thiamethoxam. Cytochrome P450 monooxygenase and carboxylesterase appear to be responsible for the resistance. Reasonable resistance management that avoids the use of cross-resistance insecticides may delay the development of resistance to thiamethoxam in this species.
|
Insecticidal activity against B-biotype thiamethoxam-sensitive Bemisia tabaci (sweet potato whitefly) in cabbage leaf assessed as mortality after 48 hr by leaf disk assay
|
Bemisia tabaci
|
1.98
ug.mL-1
|
|
Journal : Pest Manag Sci
Title : Cross-resistance study and biochemical mechanisms of thiamethoxam resistance in B-biotype Bemisia tabaci (Hemiptera: Aleyrodidae).
Year : 2010
Volume : 66
Issue : 3
First Page : 313
Last Page : 318
Authors : Feng Y, Wu Q, Wang S, Chang X, Xie W, Xu B, Zhang Y.
Abstract : BACKGROUND: B-biotype Bemisia tabaci (Gennadius) has invaded China over the past two decades. To understand the risks and to determine possible mechanisms of resistance to thiamethoxam in B. tabaci, a resistant strain was selected in the laboratory. Cross-resistance and the biochemical mechanisms of thiamethoxam resistance were investigated in the present study. RESULTS: A 66.3-fold thiamethoxam-resistant B. tabaci strain (TH-R) was established after selection for 36 generations. Compared with the susceptible strain (TH-S), the selected TH-R strain showed obvious cross-resistance to imidacloprid (47.3-fold), acetamiprid (35.8-fold), nitenpyram (9.99-fold), abamectin (5.33-fold) and carbosulfan (4.43-fold). No cross-resistance to fipronil, chlorpyrifos or deltamethrin was seen. Piperonyl butoxide (PBO) and triphenyl phosphate (TPP) exhibited significant synergism on thiamethoxam effects in the TH-R strain (3.14- and 2.37-fold respectively). However, diethyl maleate (DEM) did not act synergistically with thiamethoxam. Biochemical assays showed that cytochrome P450 monooxygenase activities increased 1.21- and 1.68-fold respectively, and carboxylesterase activity increased 2.96-fold in the TH-R strain. However, no difference was observed for glutathione S-transferase between the two strains. CONCLUSION: B-biotype B. tabaci develops resistance to thiamethoxam. Cytochrome P450 monooxygenase and carboxylesterase appear to be responsible for the resistance. Reasonable resistance management that avoids the use of cross-resistance insecticides may delay the development of resistance to thiamethoxam in this species.
|
Insecticidal activity against Drosophila suzukii assessed as male mortality at 3.31 g/ha by direct application at 22 degC measured after 24 hr
|
Drosophila suzukii
|
None
|
|
Journal : Pest Manag Sci
Title : Laboratory and field comparisons of insecticides to reduce infestation of Drosophila suzukii in berry crops.
Year : 2011
Volume : 67
Issue : 11
First Page : 1375
Last Page : 1385
Authors : Bruck DJ, Bolda M, Tanigoshi L, Klick J, Kleiber J, DeFrancesco J, Gerdeman B, Spitler H.
Abstract : BACKGROUND: The spotted wing Drosophila, Drosophila suzukii (Matsumura, 1931) (Diptera: Drosophilidae), is an invasive pest of small-fruit crops. Unlike most other Drosophila, this insect is able to oviposit into and damage ripe and ripening fruit, making it unmarketable. Because this is a new pest in the United States, it is necessary to identify registered insecticides to manage this insect effectively in conventional and organic production systems. RESULTS: The present laboratory bioassays and field trials identified a number of insecticides representing various modes of action that are effective in controlling D. suzukii. Products that performed well in the laboratory bioassay also performed well in the field, indicating that screening of new chemistries in the laboratory is a worthy exercise. Field application of pyrethoids, organophosphates or spinosyns provided 5-14 days of residual control of D. suzukii. The efficacy of the neonicotinoids as adulticides was not satisfactory compared with the other contact-mode-of-action chemistries. Based on the zero tolerance by the small-fruit industry and the individual effects mentioned above, neonicotinoids are not currently recommended for D. suzukii management. CONCLUSIONS: There are effective insecticides registered for controlling D. suzukii infestations in susceptible small-fruit crops.
|
Insecticidal activity against Drosophila suzukii assessed as male mortality at 41 g/ha by direct application at 22 degC measured after 24 hr
|
Drosophila suzukii
|
None
|
|
Journal : Pest Manag Sci
Title : Laboratory and field comparisons of insecticides to reduce infestation of Drosophila suzukii in berry crops.
Year : 2011
Volume : 67
Issue : 11
First Page : 1375
Last Page : 1385
Authors : Bruck DJ, Bolda M, Tanigoshi L, Klick J, Kleiber J, DeFrancesco J, Gerdeman B, Spitler H.
Abstract : BACKGROUND: The spotted wing Drosophila, Drosophila suzukii (Matsumura, 1931) (Diptera: Drosophilidae), is an invasive pest of small-fruit crops. Unlike most other Drosophila, this insect is able to oviposit into and damage ripe and ripening fruit, making it unmarketable. Because this is a new pest in the United States, it is necessary to identify registered insecticides to manage this insect effectively in conventional and organic production systems. RESULTS: The present laboratory bioassays and field trials identified a number of insecticides representing various modes of action that are effective in controlling D. suzukii. Products that performed well in the laboratory bioassay also performed well in the field, indicating that screening of new chemistries in the laboratory is a worthy exercise. Field application of pyrethoids, organophosphates or spinosyns provided 5-14 days of residual control of D. suzukii. The efficacy of the neonicotinoids as adulticides was not satisfactory compared with the other contact-mode-of-action chemistries. Based on the zero tolerance by the small-fruit industry and the individual effects mentioned above, neonicotinoids are not currently recommended for D. suzukii management. CONCLUSIONS: There are effective insecticides registered for controlling D. suzukii infestations in susceptible small-fruit crops.
|
Insecticidal activity against Drosophila suzukii assessed as male mortality at 112.3 g/ha by direct application at 22 degC measured after 24 hr
|
Drosophila suzukii
|
None
|
|
Journal : Pest Manag Sci
Title : Laboratory and field comparisons of insecticides to reduce infestation of Drosophila suzukii in berry crops.
Year : 2011
Volume : 67
Issue : 11
First Page : 1375
Last Page : 1385
Authors : Bruck DJ, Bolda M, Tanigoshi L, Klick J, Kleiber J, DeFrancesco J, Gerdeman B, Spitler H.
Abstract : BACKGROUND: The spotted wing Drosophila, Drosophila suzukii (Matsumura, 1931) (Diptera: Drosophilidae), is an invasive pest of small-fruit crops. Unlike most other Drosophila, this insect is able to oviposit into and damage ripe and ripening fruit, making it unmarketable. Because this is a new pest in the United States, it is necessary to identify registered insecticides to manage this insect effectively in conventional and organic production systems. RESULTS: The present laboratory bioassays and field trials identified a number of insecticides representing various modes of action that are effective in controlling D. suzukii. Products that performed well in the laboratory bioassay also performed well in the field, indicating that screening of new chemistries in the laboratory is a worthy exercise. Field application of pyrethoids, organophosphates or spinosyns provided 5-14 days of residual control of D. suzukii. The efficacy of the neonicotinoids as adulticides was not satisfactory compared with the other contact-mode-of-action chemistries. Based on the zero tolerance by the small-fruit industry and the individual effects mentioned above, neonicotinoids are not currently recommended for D. suzukii management. CONCLUSIONS: There are effective insecticides registered for controlling D. suzukii infestations in susceptible small-fruit crops.
|
Insecticidal activity against Drosophila suzukii assessed as male mortality at 2.3 g/ha by direct application at 22 degC measured after 24 hr
|
Drosophila suzukii
|
None
|
|
Journal : Pest Manag Sci
Title : Laboratory and field comparisons of insecticides to reduce infestation of Drosophila suzukii in berry crops.
Year : 2011
Volume : 67
Issue : 11
First Page : 1375
Last Page : 1385
Authors : Bruck DJ, Bolda M, Tanigoshi L, Klick J, Kleiber J, DeFrancesco J, Gerdeman B, Spitler H.
Abstract : BACKGROUND: The spotted wing Drosophila, Drosophila suzukii (Matsumura, 1931) (Diptera: Drosophilidae), is an invasive pest of small-fruit crops. Unlike most other Drosophila, this insect is able to oviposit into and damage ripe and ripening fruit, making it unmarketable. Because this is a new pest in the United States, it is necessary to identify registered insecticides to manage this insect effectively in conventional and organic production systems. RESULTS: The present laboratory bioassays and field trials identified a number of insecticides representing various modes of action that are effective in controlling D. suzukii. Products that performed well in the laboratory bioassay also performed well in the field, indicating that screening of new chemistries in the laboratory is a worthy exercise. Field application of pyrethoids, organophosphates or spinosyns provided 5-14 days of residual control of D. suzukii. The efficacy of the neonicotinoids as adulticides was not satisfactory compared with the other contact-mode-of-action chemistries. Based on the zero tolerance by the small-fruit industry and the individual effects mentioned above, neonicotinoids are not currently recommended for D. suzukii management. CONCLUSIONS: There are effective insecticides registered for controlling D. suzukii infestations in susceptible small-fruit crops.
|
Insecticidal activity against Drosophila suzukii assessed as mortality at 112.3 g/ha by direct application at 22 degC measured after 24 hr
|
Drosophila suzukii
|
None
|
|
Journal : Pest Manag Sci
Title : Laboratory and field comparisons of insecticides to reduce infestation of Drosophila suzukii in berry crops.
Year : 2011
Volume : 67
Issue : 11
First Page : 1375
Last Page : 1385
Authors : Bruck DJ, Bolda M, Tanigoshi L, Klick J, Kleiber J, DeFrancesco J, Gerdeman B, Spitler H.
Abstract : BACKGROUND: The spotted wing Drosophila, Drosophila suzukii (Matsumura, 1931) (Diptera: Drosophilidae), is an invasive pest of small-fruit crops. Unlike most other Drosophila, this insect is able to oviposit into and damage ripe and ripening fruit, making it unmarketable. Because this is a new pest in the United States, it is necessary to identify registered insecticides to manage this insect effectively in conventional and organic production systems. RESULTS: The present laboratory bioassays and field trials identified a number of insecticides representing various modes of action that are effective in controlling D. suzukii. Products that performed well in the laboratory bioassay also performed well in the field, indicating that screening of new chemistries in the laboratory is a worthy exercise. Field application of pyrethoids, organophosphates or spinosyns provided 5-14 days of residual control of D. suzukii. The efficacy of the neonicotinoids as adulticides was not satisfactory compared with the other contact-mode-of-action chemistries. Based on the zero tolerance by the small-fruit industry and the individual effects mentioned above, neonicotinoids are not currently recommended for D. suzukii management. CONCLUSIONS: There are effective insecticides registered for controlling D. suzukii infestations in susceptible small-fruit crops.
|
Displacement of [3H]alpha-BGT from nAChR in Torpedo nobiliana electric organs membranes at 300 uM by scintillation counting method
|
Torpedo nobiliana
|
12.6
%
|
|
Journal : J Pesticide Sci
Title : Pharmacological Effects of Imidacloprid and Its Related Compounds on the Nicotinic Acetyicholine Receptor with Its Ion Channel from the Torpedo Electric Organ
Year : 1995
Volume : 20
Issue : 1
First Page : 49
Last Page : 56
Authors : TOMIZAWA M, OTSUKA H, MIYAMOTO T, YAMAMOTO I
|
Displacement of [3H]PCP from nAChR in Torpedo nobiliana electric organs membranes assessed as [3H]PCP binding level at 300 uM pre-treated with 0.5 uM alpha-bungarotoxin by scintillation counting method relative to untreated control
|
Torpedo nobiliana
|
104.0
%
|
|
Journal : J Pesticide Sci
Title : Pharmacological Effects of Imidacloprid and Its Related Compounds on the Nicotinic Acetyicholine Receptor with Its Ion Channel from the Torpedo Electric Organ
Year : 1995
Volume : 20
Issue : 1
First Page : 49
Last Page : 56
Authors : TOMIZAWA M, OTSUKA H, MIYAMOTO T, YAMAMOTO I
|
Displacement of [3H]PCP from nAChR in Torpedo nobiliana electric organs membranes assessed as [3H]PCP binding level at 300 uM in presence of 100 uM carbachol by scintillation counting method relative to untreated control
|
Torpedo nobiliana
|
93.3
%
|
|
Journal : J Pesticide Sci
Title : Pharmacological Effects of Imidacloprid and Its Related Compounds on the Nicotinic Acetyicholine Receptor with Its Ion Channel from the Torpedo Electric Organ
Year : 1995
Volume : 20
Issue : 1
First Page : 49
Last Page : 56
Authors : TOMIZAWA M, OTSUKA H, MIYAMOTO T, YAMAMOTO I
|
Displacement of [3H]PCP from nAChR in Torpedo nobiliana electric organs membranes assessed as [3H]PCP binding level at 300 uM by scintillation counting method relative to untreated control
|
Torpedo nobiliana
|
221.0
%
|
|
Journal : J Pesticide Sci
Title : Pharmacological Effects of Imidacloprid and Its Related Compounds on the Nicotinic Acetyicholine Receptor with Its Ion Channel from the Torpedo Electric Organ
Year : 1995
Volume : 20
Issue : 1
First Page : 49
Last Page : 56
Authors : TOMIZAWA M, OTSUKA H, MIYAMOTO T, YAMAMOTO I
|
Displacement of [3H]alpha-BGT from nAChR in Torpedo nobiliana electric organs membranes by scintillation counting method
|
Torpedo nobiliana
|
300000.0
nM
|
|
Journal : J Pesticide Sci
Title : Pharmacological Effects of Imidacloprid and Its Related Compounds on the Nicotinic Acetyicholine Receptor with Its Ion Channel from the Torpedo Electric Organ
Year : 1995
Volume : 20
Issue : 1
First Page : 49
Last Page : 56
Authors : TOMIZAWA M, OTSUKA H, MIYAMOTO T, YAMAMOTO I
|
Displacement of [3H]alpha-BGT from nAChR in Apis mellifera (honeybee) head homogenates
|
Apis mellifera
|
8830.0
nM
|
|
Journal : J Pesticide Sci
Title : Pharmacological Characteristics of Insect Nicotinic Acetyicholine Receptor with Its Ion Channel and the Comparison of the Effect of Nicotinoids and Neonicotinoids
Year : 1995
Volume : 20
Issue : 1
First Page : 57
Last Page : 64
Authors : TOMIZAWA M, OTSUKA H, MIYAMOTO T, ELDEFRAWI ME, YAMAMOTO I
|
Displacement of [3H]PCP from nAChR in Apis mellifera (honeybee0 head homogenates
|
Apis mellifera
|
277000.0
nM
|
|
Journal : J Pesticide Sci
Title : Pharmacological Characteristics of Insect Nicotinic Acetyicholine Receptor with Its Ion Channel and the Comparison of the Effect of Nicotinoids and Neonicotinoids
Year : 1995
Volume : 20
Issue : 1
First Page : 57
Last Page : 64
Authors : TOMIZAWA M, OTSUKA H, MIYAMOTO T, ELDEFRAWI ME, YAMAMOTO I
|
Resistance factor, LC50 for Frankliniella occidentalis R1S (western flower thrips) to LC50 for Frankliniella occidentalis TFR
|
Frankliniella occidentalis
|
0.2
|
|
Journal : Pest Manag Sci
Title : Thiamethoxam acts as a target-site synergist of spinosad in resistant strains of Frankliniella occidentalis.
Year : 2013
Volume : 69
Issue : 2
First Page : 188
Last Page : 194
Authors : Guillén J, Bielza P.
Abstract : Previous studies have suggested that the resistance mechanism towards spinosad in Frankliniella occidentalis (Pergande) is an altered target site. Like the neonicotinoids, the spinosyns act on nicotinic acetylcholine receptors (nAChRs) in insects, but at a distinct site. The changes in nAChRs related to spinosad resistance in thrips might involve interaction with neonicotinoids. In this study, the efficacy of spinosad and neonicotinoids, alone and in combination, was evaluated in susceptible and spinosad-resistant thrips strains.The neonicotinoids tested were imidacloprid, thiacloprid, acetamiprid, thiamethoxam and clothianidin. No cross-resistance was shown between spinosad and any of the neonicotinoids. However, an increased toxicity was observed when a mixture of spinosad with thiamethoxam or clothianidin was tested. No synergism was found in the susceptible strains. The more spinosad-resistant the thrips strain, the stronger was the synergism.Data suggest that spinosad and thiamethoxam may interact at the nAChRs in spinosad-resistant thrips, facilitating enhanced insecticidal action.
|
Resistance factor, LC50 for Frankliniella occidentalis PPP (western flower thrips) to LC50 for Frankliniella occidentalis TFR
|
Frankliniella occidentalis
|
0.2
|
|
Journal : Pest Manag Sci
Title : Thiamethoxam acts as a target-site synergist of spinosad in resistant strains of Frankliniella occidentalis.
Year : 2013
Volume : 69
Issue : 2
First Page : 188
Last Page : 194
Authors : Guillén J, Bielza P.
Abstract : Previous studies have suggested that the resistance mechanism towards spinosad in Frankliniella occidentalis (Pergande) is an altered target site. Like the neonicotinoids, the spinosyns act on nicotinic acetylcholine receptors (nAChRs) in insects, but at a distinct site. The changes in nAChRs related to spinosad resistance in thrips might involve interaction with neonicotinoids. In this study, the efficacy of spinosad and neonicotinoids, alone and in combination, was evaluated in susceptible and spinosad-resistant thrips strains.The neonicotinoids tested were imidacloprid, thiacloprid, acetamiprid, thiamethoxam and clothianidin. No cross-resistance was shown between spinosad and any of the neonicotinoids. However, an increased toxicity was observed when a mixture of spinosad with thiamethoxam or clothianidin was tested. No synergism was found in the susceptible strains. The more spinosad-resistant the thrips strain, the stronger was the synergism.Data suggest that spinosad and thiamethoxam may interact at the nAChRs in spinosad-resistant thrips, facilitating enhanced insecticidal action.
|
Insecticidal activity against Frankliniella occidentalis R1S (western flower thrips) in compound treated sweet pepper leaf assessed as mortality at 25 +/- 2 degC by leaf dip method
|
Frankliniella occidentalis
|
119.0
ug.mL-1
|
|
Journal : Pest Manag Sci
Title : Thiamethoxam acts as a target-site synergist of spinosad in resistant strains of Frankliniella occidentalis.
Year : 2013
Volume : 69
Issue : 2
First Page : 188
Last Page : 194
Authors : Guillén J, Bielza P.
Abstract : Previous studies have suggested that the resistance mechanism towards spinosad in Frankliniella occidentalis (Pergande) is an altered target site. Like the neonicotinoids, the spinosyns act on nicotinic acetylcholine receptors (nAChRs) in insects, but at a distinct site. The changes in nAChRs related to spinosad resistance in thrips might involve interaction with neonicotinoids. In this study, the efficacy of spinosad and neonicotinoids, alone and in combination, was evaluated in susceptible and spinosad-resistant thrips strains.The neonicotinoids tested were imidacloprid, thiacloprid, acetamiprid, thiamethoxam and clothianidin. No cross-resistance was shown between spinosad and any of the neonicotinoids. However, an increased toxicity was observed when a mixture of spinosad with thiamethoxam or clothianidin was tested. No synergism was found in the susceptible strains. The more spinosad-resistant the thrips strain, the stronger was the synergism.Data suggest that spinosad and thiamethoxam may interact at the nAChRs in spinosad-resistant thrips, facilitating enhanced insecticidal action.
|
Insecticidal activity against Frankliniella occidentalis PPP (western flower thrips) in compound treated sweet pepper leaf assessed as mortality at 25 +/- 2 degC by leaf dip method
|
Frankliniella occidentalis
|
116.0
ug.mL-1
|
|
Journal : Pest Manag Sci
Title : Thiamethoxam acts as a target-site synergist of spinosad in resistant strains of Frankliniella occidentalis.
Year : 2013
Volume : 69
Issue : 2
First Page : 188
Last Page : 194
Authors : Guillén J, Bielza P.
Abstract : Previous studies have suggested that the resistance mechanism towards spinosad in Frankliniella occidentalis (Pergande) is an altered target site. Like the neonicotinoids, the spinosyns act on nicotinic acetylcholine receptors (nAChRs) in insects, but at a distinct site. The changes in nAChRs related to spinosad resistance in thrips might involve interaction with neonicotinoids. In this study, the efficacy of spinosad and neonicotinoids, alone and in combination, was evaluated in susceptible and spinosad-resistant thrips strains.The neonicotinoids tested were imidacloprid, thiacloprid, acetamiprid, thiamethoxam and clothianidin. No cross-resistance was shown between spinosad and any of the neonicotinoids. However, an increased toxicity was observed when a mixture of spinosad with thiamethoxam or clothianidin was tested. No synergism was found in the susceptible strains. The more spinosad-resistant the thrips strain, the stronger was the synergism.Data suggest that spinosad and thiamethoxam may interact at the nAChRs in spinosad-resistant thrips, facilitating enhanced insecticidal action.
|
Insecticidal activity against Frankliniella occidentalis TFB (western flower thrips) in compound treated sweet pepper leaf assessed as mortality at 25 +/- 2 degC by leaf dip method
|
Frankliniella occidentalis
|
663.0
ug.mL-1
|
|
Journal : Pest Manag Sci
Title : Thiamethoxam acts as a target-site synergist of spinosad in resistant strains of Frankliniella occidentalis.
Year : 2013
Volume : 69
Issue : 2
First Page : 188
Last Page : 194
Authors : Guillén J, Bielza P.
Abstract : Previous studies have suggested that the resistance mechanism towards spinosad in Frankliniella occidentalis (Pergande) is an altered target site. Like the neonicotinoids, the spinosyns act on nicotinic acetylcholine receptors (nAChRs) in insects, but at a distinct site. The changes in nAChRs related to spinosad resistance in thrips might involve interaction with neonicotinoids. In this study, the efficacy of spinosad and neonicotinoids, alone and in combination, was evaluated in susceptible and spinosad-resistant thrips strains.The neonicotinoids tested were imidacloprid, thiacloprid, acetamiprid, thiamethoxam and clothianidin. No cross-resistance was shown between spinosad and any of the neonicotinoids. However, an increased toxicity was observed when a mixture of spinosad with thiamethoxam or clothianidin was tested. No synergism was found in the susceptible strains. The more spinosad-resistant the thrips strain, the stronger was the synergism.Data suggest that spinosad and thiamethoxam may interact at the nAChRs in spinosad-resistant thrips, facilitating enhanced insecticidal action.
